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Computer Science: Computer hardware - 0 views

computersciencetopper.blogspot.com/...computer-hardware.html Best computer computer computer Science it blog
shared by umar111 on 26 Apr 23 - No Cached
  • umar111 on 26 Apr 23
     
    Computer Science Tuesday, April 25, 2023 Computer hardware Computer hardware is the physical components that make up a computer system. It includes everything from the central processing unit (CPU) to the monitor, keyboard, and mouse. Understanding the different types of hardware and how they work together is essential for anyone who works with computers. In this article, we will explore the various components of computer hardware, including internal and external components, and the peripherals that connect to them. We will also discuss the importance of hardware maintenance, the latest advancements in computer technology, and factors to consider when choosing the right hardware for your needs. Whether you are a computer technician, a gamer, or simply someone who uses a computer for everyday tasks, this article will help you better understand the world of computer hardware. Introduction to Computer Hardware Computer hardware refers to the physical components that make up a computer system. It includes everything from the processor and memory to input/output devices such as the keyboard and monitor. In this article, we will explore the different types of computer hardware and their functions. What is Computer Hardware Computer hardware refers to the physical components of a computer system. It includes all the components that can be touched, seen, and used to interact with a computer, such as the monitor, keyboard, and mouse. Hardware is different from computer software, which refers to the programs and applications that run on a computer system. History of Computer Hardware The history of computer hardware dates back to the 1820s when Charles Babbage, an English mathematician, and inventor, designed the first analytical engine, which was considered to be the first mechanical computer. With time, more complex electronic computers were developed, including the first Intel microprocessor in 1971. Since then, computer hardware has continued to evolve, becoming
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Low-dose penicillin in early life induces long-term changes in murine gut microbiota, b... - 0 views

www.nature.com/...ncomms15062 brain brain health probiotics antibiotics gut-brain gut bacteria gut flora gut health gut microbiome gut microbiota gut
shared by Nathan Goodyear on 26 Oct 17 - No Cached
  • Nathan Goodyear on 26 Oct 17
     
    Gut bacteria brain connection: animal study finds antibiotics early in life alter gut flora which effects young brain and alters behavior; probiotics with Lactobacillus rhamnosus JB-1 appears to blunt the altered Gut bacteria brain connection.
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Windows Technical Support Number 1 800 261 4071 - 0 views

www.postaddz.com/...ort_number_1_800_261_4071.html Windows technical customer services number support
shared by Windows Customer Support Number on 26 Jul 16 - No Cached
  • Windows Customer Support Number on 26 Jul 16
     
    Windows support number 1-800-261-4071 is a staff of best technicians and experts who are will to help our clients at any time. We provide our services over the phone for common issues and for complicated issues we conduct a remote connection technology. just dial our Windows technical support number 1-800-261-4071 for an urgent need and help, you can also visit our websites as http://technicalsupportnumberforwindows.com
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Windows technical support number 1-800-261-4071 - 0 views

www.trustlink.org/...umber-1-800-261-4071-207112608 Windows technical computer support services customer
shared by Windows Customer Support Number on 13 Jun 16 - No Cached
  • Windows Customer Support Number on 13 Jun 16
     
    Levon tech services are a big company of more than 100 certified and experienced technicians, we are always available during the weekend or on holidays.We provide our services over the phone for common issues and for complicated issues we conduct a remote connection technology. Just pick up your phone and dial our Windows technical support number 1-800-261-4071
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Windows tech support number 1-800-261-4071 - 0 views

www.youtube.com/watch Windows technical customer services technical-support help
shared by Windows Customer Support Number on 09 Jul 16 - No Cached
  • Windows Customer Support Number on 09 Jul 16
     
    Having any issues With your Windows software, just dial our Windows support number 1-800-261-4071 for all the Windows software related issues,We will connect you with a certified technician and ensue that you get a speedy and effective solution to your worries,
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Windows support number 1-800-261-4071 - 0 views

www.dailymotion.com/x4jynj1 Windows technical customer services tech support
shared by Windows Customer Support Number on 11 Jul 16 - No Cached
  • Windows Customer Support Number on 11 Jul 16
     
    Windows Technical Support Number 1-800-261-4071 we are a big company of more than 100 certified and experienced technicians. We provide our services over the phone for common issues and we conduct a remote connection technology also. We solve all our customers issues solution without wasting any time. just call us on our Windows technical support number 1-800-261-4071, or you can also go through our websites as http://technicalsupportnumberforwindows.com
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Posttraumatic Stress Disorder: Does the Gut Microbiome Hold the Key? - 0 views

cpa.sagepub.com/...204.abstract stress PTSD post traumatic stress disorder gut health gut flora gut gut microbiome gut microbiota
shared by Nathan Goodyear on 04 May 16 - No Cached
  • Nathan Goodyear on 04 May 16
     
    Gut microbiome diversity is different in stressed mice and calm mice in animal study.  Authors propose intricate connection between gut:brain and possible evaluation and therapy in PTSD.
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Colonic CD8 and gamma delta T-cell infiltration with epithelial damage in children with... - 0 views

www.ncbi.nlm.nih.gov/...11241044 ASD autism inflammation children Lymphoid nodular hyperplasia LNH regressive
shared by Nathan Goodyear on 30 Jan 12 - No Cached
  • Nathan Goodyear on 30 Jan 12
     
    Study shows that gastrointestinal epithelial dysfunction is found in autism. This would play a critical role in systemic inflammation and would contribute to ASD symptoms through the gut-brain connection.
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short-term benefit from oral vancomycin treatment of regressive-onset autism - 0 views

www.ncbi.nlm.nih.gov/pubmed autism ASD dysbiosis inflammation gut GI gastrointestinal children
shared by Nathan Goodyear on 30 Jan 12 - No Cached
  • Nathan Goodyear on 30 Jan 12
     
    disruption of gut bacteria balance, dysbiosis, proposed to play role in regression autism. This study treated these children with vancomycin. Short interval improvement was found. The take home from this study is the gut-brain connection in children with regression autism
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Dysregulated Innate Immune Responses in Young Children with Autism Spectrum Disorders: ... - 0 views

mercola.fileburst.com/...europsych%20--%20Jyonouchi.pdf autism ASD LPS inflammation innate immune system gut gastrointestinal
shared by Nathan Goodyear on 29 Jan 12 - No Cached
  • Nathan Goodyear on 29 Jan 12
     
    Dysfunctions of the innate immune system, in the gastrointestinal system, in children with autism found. LPS found to be involved. Again, revealing a gut-brain connection in autism.
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The Complex Role of Estrogens in Inflammation - 0 views

edrv.endojournals.org/...521.full estrogens inflammation
shared by Nathan Goodyear on 14 Aug 12 - No Cached
  • These studies suggest inflammation-dependent up-regulation of ERβ relative to ERα.
  • up-regulation of ERβ relative to ERα under hypoxic conditions, which might lead to a preponderance of signaling through ERβ pathways
  • it seems that E2 at periovulatory to pregnancy levels inhibited proinflammatory cytokines from PBMCs
  • ...26 more annotations...
  • it is clear that E2 can stimulate antibody production by B cells, probably by inhibiting T cell suppression of B cells
  • In cycling women, the largest quantities of Ig were detected before ovulation
  • In contrast, E2 at high concentrations leads to a suppression of B-lymphocyte lineage precursors
  • E2 at periovulatory to pregnancy serum levels is able to stimulate antibody secretion under healthy conditions but also in autoimmune diseases, whereas similar serum levels of E2 lead to a suppression of bone marrow B cell lineage precursors
  • In chronic inflammatory disorders, where B cells play a decisive role, E2 would promote the disease when autoaggressive B cells are already present, whereas chronically elevated E2 would inhibit initiation of an autoimmune disease when no such B cells are available. This might be a good reason why particularly B cell-dependent diseases such as SLE, mixed connective tissue disease (Sharp syndrome), IgA nephropathy, dermatitis herpetiformis, gluten sensitive enteropathy, myasthenia gravis, and thyroiditis appear in women in the reproductive years, predominantly, in the third or fourth decades of life
  • Th17 cells are thought to be the main responsible cells for chronic inflammatory tissue destruction in autoimmune diseases
  • IFN-γ, IL-12, and TNF were allocated to Th1 reactions
  • IL-4, IL-5, and IL-10 to Th2 responses
  • antiinflammatory T regulatory cells producing TGF-β and proinflammatory T helper type 17 cells (Th17) producing IL-17
  • no direct effects of estrogens on Th17 cells or IL-17 secretion have been described until now.
  • So-called Th17 cells producing IL-17 are the main T cells responsible for chronic inflammation.
  • Because IFN-γ has been allocated a Th17-inhibiting role (Fig. 1⇑), its increase by E2 at pregnancy doses and the E2-mediated inhibition of TNF must be viewed as a favorable effect in chronic inflammation
  • in humans and mice, E2 at periovulatory to pregnancy levels stimulates IL-4, IL-10, and IFN-γ but inhibits TNF from CD4+ T cells
  • In humans and mice, E3 and E2, respectively, at pregnancy levels inhibit T cell-dependent delayed type hypersensitivity
  • increased IL-4, IL-10, and IFN-γ in the presence of low TNF support an antiaggressive immune response
  • secretion of IL-1β is increased at periovulatory/proestrus to early pregnancy levels, whereas IL-1 secretion is inhibited at high pregnancy levels
  • The dichotomous effect of E2 on IL-1β and TNF at high and low concentrations is most probably due to inhibition of NF-κB at high concentrations
  • experiments with mouse and rat macroglial and microglial cells demonstrate that E2 at proestrus to pregnancy levels exerts neuroprotective effects by increasing TGF-β and by inhibiting iNOS and NO release, and reducing expression of proinflammatory cytokines and prostaglandin E2 production.
  • E2 at periovulatory to pregnancy levels inhibits NF-κB activation, which must be viewed as an antiinflammatory signal
  • It was shown that E2 concentrations equal to or above 10−10 m are necessary to inhibit NF-κB activation
  • important proinflammatory cytokines are typically inhibited at periovulatory (proestrus) to pregnancy levels of E2, which is evident for IL-6, IL-8, and TNF
  • low E2 concentrations were demonstrated to have no or even stimulatory effects
  • This renders a woman in the postmenopausal phase to a more proinflammatory situation
  • most in vitro studies demonstrated a stimulatory effect of E2 on secretion of IL-4, IL-10, and TGF-β typically at periovulatory to pregnancy levels
  • E2 at periovulatory to pregnancy levels has an ameliorating effect on chronic inflammatory diseases as long as B cell-dependent immunity or an overshooting fibrotic tissue repair process do not play a crucial pathogenic role. However, when the B cell plays an important role, E2 might even stimulate the disease process as substantiated by flare-ups in SLE during pregnancy
    • Nathan Goodyear
      Nathan Goodyear on 15 Aug 12
       
      SLE, mixed connective tissue disease (Sharp syndrome), IgA nephropathy, dermatitis herpetiformis, gluten sensitive enteropathy, myasthenia gravis, and thyroiditis
  • Short-term administration of E2 at pregnancy levels was shown to induce an inflammatory response specific to the lateral prostate of the castrated male rat
  • Nathan Goodyear on 14 Aug 12
     
    great review of the complex interaction between Estrogens and inflammation.  Reference here is in females.
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Stress in obesity: cause or consequence? [Med Hypotheses. 2011] - PubMed - NCBI - 0 views

www.ncbi.nlm.nih.gov/...21444159 stress obesity overweight weight-gain weight loss cortisol
shared by Nathan Goodyear on 10 Feb 12 - No Cached
  • Nathan Goodyear on 10 Feb 12
     
    stress and obesity.  An intimate connection.
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The role of gut microbiota in the gut-brain axis: current challenges and perspectives. ... - 0 views

www.researchgate.net/...nt_challenges_and_perspectives gut brain microbiota dysbiosis gut-brain axis
shared by Nathan Goodyear on 17 Feb 14 - No Cached
  • Nathan Goodyear on 17 Feb 14
     
    Great discussion of the gut brain connection.
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Oxytocin during the initial stages of romantic attachment: Relations to couples' intera... - 0 views

www.ncbi.nlm.nih.gov/...PMC3936960 oxytocin
shared by Nathan Goodyear on 19 Mar 14 - No Cached
  • Nathan Goodyear on 19 Mar 14
     
    Oxytocin levels linked to a couples romantic connection, not sex.
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Testosterone deficiency: a key factor in the... [J Womens Health. 1998] - PubMed - NCBI - 0 views

www.ncbi.nlm.nih.gov/...9785308 Testosterone women CVD cardiovascular disease hysterectomy hormone hormones post-menopausal
shared by Nathan Goodyear on 21 Jul 13 - No Cached
  • Nathan Goodyear on 21 Jul 13
     
    Weak connection, but this study finds that Testosterone in post-menopausal women will reduce cardiovascular disease (CVD) in women.  Their conclusion is based on the rise in CVD in post-menopausal women and the decline in Testosterone levels post-hysterectomy. That is the one instant where Testosterone levels do precipitously decline.  Contrast this with natural menopause where Testosterone does not appear to decline as precipitously.
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Slow CCL2-dependent translocation of biopersistent particles from muscle to brain - 0 views

www.ncbi.nlm.nih.gov/...PMC3616851 vaccines vaccinations aluminum AL brain
shared by Nathan Goodyear on 01 Oct 14 - No Cached
  • Nathan Goodyear on 01 Oct 14
     
    Study finds translocation of alum from vaccines from muscle to brain in some individuals.  This leads to the neurologically condition termed Macrophagic myofasciitis.  Study after study has shown this connection and now studies are showing this translocation of immune cells to the brain resulting in neuro-inflammation.  All I hear from pro vaccinators is vitriol.  Here is science.
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Dietary intervention impact on gut microbial gene richness : Nature : Nature Publishing... - 0 views

www.nature.com/...nature12480.html gut bacteria dysbiosis obesity overweight diet nutrition metabolism metabolic endotoxemia
shared by Nathan Goodyear on 29 Aug 13 - No Cached
  • Nathan Goodyear on 29 Aug 13
     
    Nutrition is a language.  Poor gut balance and as what the authors call "richness" contributes to metabolic dysfunction and obesity.  This study found that Nutrition can improve gut bacterial balance and "richness" that then will improve the metabolic impact of the nutrition. Complex and beautiful nutrition--gut--metabolism connection.
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Gut-brain link grabs neuroscientists : Nature News & Comment - 0 views

www.nature.com/...-grabs-neuroscientists-1.16316 gut flora gut gut microbiome gut microbiota gut-brain autism
shared by Nathan Goodyear on 29 Jun 15 - No Cached
  • Nathan Goodyear on 29 Jun 15
     
    Gut-brain connection via gut microbiome starts to move mainstream.
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Buy IV Supplies & Refurbished Medical Equipment - 0 views

www.zipleaf.us/...Willow-Medical_33727 IV Supplies Refurbished Medical Equipment
shared by Willow O'Donnell on 17 Apr 15 - No Cached
  • Willow O'Donnell on 17 Apr 15
     
    Willow Med connected by Willow Medical. It's provides the best medical equipment at affordable prices, Inc. are Medical/Safety Supplies, IV pumps, IV Medical Supplies & Refurbished Medical Equipment to various market segments.
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Dietary fat and mammary cancer. I. Promoting effec... [J Natl Cancer Inst. 1986] - PubM... - 0 views

www.ncbi.nlm.nih.gov/...3459924 dietary fat cancer omega-6
shared by Nathan Goodyear on 10 Feb 11 - No Cached
  • Nathan Goodyear on 10 Feb 11
     
    review of 97 animal studies reveal connection between high omega-6 dietary intake and cancer growth
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