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Great review of lipid peroxidation

This study finds a strong correlation between advancing breast cancer, decreased catalase and SOD with increasing MDA.  The authors of this study conclude this is a key factor in carcinogenesis and not a by-product of cancer.  This flies in the face of traditional medicines fear of antioxidant therapy in cancer.

Great discussion on the free-radical theory of aging and the effects of mitochondria

Study finds a reduction in SuperOxide Dismutase and Glutathione Perioxidase in advancing breast cancer.  Cancer is a high oxidative stress disease that results in inflammation, mitochondrial dysfunction and proliferation.  Adenosine Deaminase (ADA) is proposed to be another biomarker to assess tumor stage.  

This study finds that AGE, advanced glycation end products, play a significant role in free radical generation and neurodegenerative disease. Quote: "... pharmacologoical approaches which break the vicious cycle of oxidative stress and neurodegeneration offer new opportunities for the treatment of AD. These approaches include AGE- inhibitors, antioxidants, and anti-inflammatory substances which prevent radical production." They focus on the pharmacological, how about the natural first?

Great read!  If you want to see how free radicals and oxidative stress contribute to inflammation and disease (DM in this case), read this article.

How free radicals lead to aging

IV Vitamin C improves endothelial vasodilation in essential hypertension.  The Vitamin C reduces the oxygen free radicals which allowed eNOS to increase NO production.  Two take homes: oxygen free radicals may be responsible for the endothelial dysfunction that leads to essential hypertension and vitamin C, particularly IV, can be used to counter this process.  Other studies have shown IV vitamin C to be anti-hypertensive in its action.

vitamin C shown to benefit stroke patients.  Vitamin C, ascorbic acid, is extremely prevalent in the brain.  Functions to scavenge free radicals

ferritin released enhanced pharmacologic ascorbate induced-cytotoxicity, indicating that ferritin with high iron-saturation could be a source of catalytic iron. Consistent with this, ascorbate has also been shown to be capable of releasing iron from cellular ferritin

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elevated insulin levels associated with disordered Beta-amyloid metabolism and hyperphosphorylation of tau proteins.  Free radicals also play a role as well.

Green tea extract, EGCG, inhibits free-radical damage

Hard to show causation here, but volume of environmental toxicants associated with shortening of Telomeres.  Mechanism?  Increased free radical induced mitochondrial disruption resulting in relocation of the Telomerase enzyme from the mitochondria to the nuclei.   This has been shown to occur in cancer pathogenesis.

vitamin C released in the brain with an ischemic stroke.  Question: is this a way to provide scavenger for free radicals following a stroke?

NAC shown to improve secondary measures of cognitive function in small study in those with Alzheimer's disease. NAC proves to be a reliable means to reduce free radical induced advanced glycation end productions and resultant inflammation. Also was shown to be well tolerated.

NAD+ is very important in ATP production and as a means to counter free radicals.  This is a nice discussion of NAD metabolism.  

curcumin functions to reduce inflammation and free radicals.  Curcumin also increases BDNF post TBI, which will improve synaptic plasticity, cognition, and memory.  

Must read on the free radical theory of aging to application of disease prevention as it relates to neurodegenerative disease