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Androgen deprivation therapy resulted in an 11% increase in fat mass and just under 4% decrease in lean muscle mass in men.  

Only abstract available here--high Estradiol:Testosterone, thus high aromatase activity, increased abdominal fat and reduced lean muscle mass.  The authors also reported an higher bone mineral density, but that pales in the comparison of the metabolic dysfunction of high fat and low muscle mass causes.

Moderate resistance training program over 8 weeks associated with increased Testosterone production in young and middle age lean men.  Growth hormone was also increased in both groups.  ACTH and cortisol decreased in this lean men.  

lean women with PCOS found to have reduced 11 beta-HSD1 and increased 5 alpha reductase activity.  This, in part, is associated with the elevated cortisol and androgens in these women.

Testosterone shown to slow loss of lean body mass, also known as muscle.

Androgen deprivation therapy was found to decrease lean muscle mass and increase abdominal adipose tissue, not visceral.  Significant change in body composition in men depleted of androgens in androgen deprivation therapy.

Chemotherapy is associated with a decline in lean body mass.  This could play a role in increase weight, especially in pre menopause women.

Rat study found that High fat and high branch chain amino acid intake long-term leads to insulin resistance.  This occurred through phosphorylation of insulin signaling downstream and differed between lean versus obese individuals.  

D-chiro-inositol reduced insulin levels and associated male hormone production in lean women with PCOS

alpha lipoic acid improves energy production, lean mass and weight loss

Study finds no improvement with glucose control in diabetics.  This study looked at moderately controlled diabetes. Studies have previously shown that poorly controlled diabetes definitely benefits more than those with more mild glucose control problems.  Additionally, the Testosterone levels in this study would not have met the definition of low T by other studies.  So, the question is did these men need T?  Second, did the authors design the study long enough to see changes in the insulin sensitivity and glucose control?  Abstract only available and thus I don't have access to that information.  Third, and this might support the 2nd point, increased lean mass and decreased fat mass was found.  This points to positive metabolic change.  Would this have, given more time, resulted in improved glucose control? No change in visceral adiposity was seen.  This finding, also, is not new.  Testosterone therapy does not improve visceral adiposity.  Though, increasing fat adiposity, low Testosterone, and associated increase in systemic inflammatory cytokine production results in visceral adiposity, Testosterone therapy does reverse the visceral adiposity.  

Meta-analysis finds that Testosterone therapy in men over 60 increased lean mass and decreased fat mass.

Great review of the Hypogonadal-obesity-adipocytokine hypothesis.

Great read on the ketogenic  and its application to sports/training...

macrophages (M1) in obese individuals are more pro-inflammatory than those in lean (M2) individuals. The exact mechanism to explain the switch from M2 to M1 has yet to be determined, but this study proposes that lipotoxic oxidized LDL is a possible target

The prevalence of low T varies significantly between lean versus overweight men.  This suggests a significant contribution of obesity and its biologic effects as a cause of low T.  The difference is 30%.