Long term nutritional intake and the risk for non-alcoholic fatty liver disease (NAFLD): a population based study.
Zelber-Sagi S, Nitzan-Kaluski D, Goldsmith R, Webb M, Blendis L, Halpern Z, Oren R.
J Hepatol. 2007 Nov;47(5):711-7. Epub 2007 Aug 14.
PMID: 17850914
doi:10.1016/j.jhep.2007.06.020
Immunologic effects of national cholesterol education panel step-2 diets with and without fish-derived N-3 fatty acid enrichment.
Meydani SN, Lichtenstein AH, Cornwall S, Meydani M, Goldin BR, Rasmussen H, Dinarello CA, Schaefer EJ.
J Clin Invest. 1993 Jul;92(1):105-13.
PMID: 8325975
doi:10.1172/JCI116537
the low-fat, high-fish diet significantly decreased the percentage of helper T cells whereas the percentage of suppressor T cells increased
Vitamin D and Cancer Mini-Symposium: The Risk of Additional Vitamin D.
Vieth R.
Ann Epidemiol. 2009 Apr 11. [Epub ahead of print]
PMID: 19364661
doi:10.1016/j.annepidem.2009.01.009
Aging decreases the capacity of human skin to produce vitamin D3.
MacLaughlin J, Holick MF.
J Clin Invest. 1985 Oct;76(4):1536-8.
PMID: 2997282
doi:10.1172/JCI112134
An evaluation of surgically obtained skin (age range, 8-92 yr) revealed that there is an age-dependent decrease in the epidermal concentrations of provitamin D3 (7-dehydrocholesterol). To ascertain that aging indeed decreased the capacity of human skin to produce vitamin D3, some of the skin samples were exposed to ultraviolet radiation and the content of previtamin D3 was determined in the epidermis and dermis. The epidermis in the young and older subjects was the major site for the formation of previtamin D3, accounting for greater than 80% of the total previtamin D3 that was produced in the skin. A comparison of the amount of previtamin D3 produced in the skin from the 8- and 18-yr-old subjects with the amount produced in the skin from the 77- and 82-yr-old subjects revealed that aging can decrease by greater than twofold the capacity of the skin to produce previtamin D3. Recognition of this difference may be extremely important for the elderly, who infrequently expose a small area of skin to sunlight and who depend on this exposure for their vitamin D nutritional needs.
Vitamin D deficiency is the cause of common obesity.
Foss YJ.
Med Hypotheses. 2009 Mar;72(3):314-21. Epub 2008 Dec 2.
PMID: 19054627
doi:10.1016/j.mehy.2008.10.005
Effect of four monthly oral vitamin D3 (cholecalciferol) supplementation on fractures and mortality in men and women living in the community: randomised double blind controlled trial.
Trivedi DP, Doll R, Khaw KT.
BMJ. 2003 Mar 1;326(7387):469.
PMID: 12609940
CONCLUSION: Four monthly supplementation with 100 000 IU oral vitamin D may prevent fractures without adverse effects in men and women living in the general community.
Geographic variation in breast cancer mortality in the United States: a hypothesis involving exposure to solar radiation.
Garland FC, Garland CF, Gorham ED, Young JF.
Prev Med. 1990 Nov;19(6):614-22.
PMID: 2263572
doi:10.1016/0091-7435(90)90058-R
Vitamin D from sunlight exposure may be associated with low risk for fatal breast cancer, and differences in ultraviolet light reaching the United States population may account for the striking regional differences in breast cancer mortality. The ecological nature of this study is emphasized, and the possibility that an indirect association with dietary and socioeconomic factors could explain these findings is discussed.
Environmental risk factors for autism: Do they help cause de novo genetic mutations that contribute to the disorder?
Kinney DK, Barch DH, Chayka B, Napoleon S, Munir KM.
Med Hypotheses. 2009 Aug 20. [Epub ahead of print]
PMID: 19699591
Vitamin D treatment in multiple sclerosis.
Myhr KM.
J Neurol Sci. 2009 Jun 22. [Epub ahead of print]
PMID: 19549608
doi:10.1016/j.jns.2009.05.002
Epidemiological evidence combined with clinical and laboratory analyses, and experimental animal models, suggest a possible influence of vitamin D on MS susceptibility as well as clinical disease activity.
Supplement with vitamin D may reduce the risk of developing MS. An intervention may also reduce the risk of conversion from a first clinical event suggestive of MS to clinical definite MS, as well as reduce the relapse rate among patients with relapsing remitting MS. More studies are, however, needed to determine optimal dose and serum level for vitamin D, as well as target populations and optimal timing for intervention.
Vitamin D, nervous system and aging.
Tuohimaa P, Keisala T, Minasyan A, Cachat J, Kalueff A.
Psychoneuroendocrinology. 2009 Aug 4. [Epub ahead of print]
PMID: 19660871
Food chain as origin of vitamin D in fish
D. Sunita Rao and N. Raghuramulu.
Comparative Biochemistry and Physiology Part A: Physiology
Volume 114, Issue 1, May 1996, Pages 15-19
doi:10.1016/0300-9629(95)02024-1
Plankton, the chief food source of fish, was assessed as the possible dietary origin of vitamin D in fish. The presence of vitamin D compounds were examined in fresh water phytoplankton and zooplankton employing a series of chromatographic procedures. Abundant amounts of provitamins D and vitamins D (D2 and D3) were found in the fresh water plankton. The high amount of vitamin D observed may be due to exposure of plankton to sunlight because the plankton were caught during the summer month. Thus, plankton may be an important contributor to vitamin D in fish.
Comparison of biochemical effects of statins and fish oil in brain: the battle of the titans.
Farooqui AA, Ong WY, Horrocks LA, Chen P, Farooqui T.
Brain Res Rev. 2007 Dec;56(2):443-71. Epub 2007 Oct 23. Review.
PMID: 17959252
doi:10.1016/j.brainresrev.2007.09.004
The contributions of aspirin and microbial oxygenase to the biosynthesis of anti-inflammatory resolvins: novel oxygenase products from omega-3 polyunsaturated fatty acids.
Arita M, Clish CB, Serhan CN.
Biochem Biophys Res Commun. 2005 Dec 9;338(1):149-57. Epub 2005 Aug 10. Review.
PMID: 16112645
doi:10.1016/j.bbrc.2005.07.181
An outbreak of hypervitaminosis D associated with the overfortification of milk from a home-delivery dairy.
Blank S, Scanlon KS, Sinks TH, Lett S, Falk H.
Am J Public Health. 1995 May;85(5):656-9.
PMID: 7733425
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