Skip to main content

Home/ Nutrition/ Group items tagged research

Rss Feed Group items tagged

Matti Narkia

Blueberry Supplementation Improves Memory in Older Adults† - Journal of Agric... - 3 views

  •  
    Blueberry Supplementation Improves Memory in Older Adults (dagger).
    Krikorian R, Shidler MD, Nash TA, Kalt W, Vinqvist-Tymchuk MR, Shukitt-Hale B, Joseph JA.
    J Agric Food Chem. 2010 Jan 4. [Epub ahead of print]
    PMID: 20047325
    DOI: 10.1021/jf9029332

    The findings of this preliminary study suggest that moderate-term blueberry supplementation can confer neurocognitive benefit and establish a basis for more comprehensive human trials to study preventive potential and neuronal mechanisms.
MrGhaz .

Learning Better Eating Habits - 1 views

  •  
    Look at your patterns of eating diary. Are you eating all over your home or at work? Do you sometimes eat standing up? Or whilst doing other things? All of these situations may trigger of eating. Try to eat in just one place at home - and one at work. Sit down at a table and don't read or watch TV. You will gradually weaken your wide range of eating triggers and only want to eat in the 'proper place'.
MrGhaz .

A Balanced Diet - 2 views

  •  
    The amount of energy to be provided by a balanced diet for any person will depend on several factors: age, sex, body size, lifestyle, occupation, and the climate of the area he is in. The proportion of the various food classes in a diet is also not the same for different persons, due to varying physical and physiological requirements..
MrGhaz .

The Importance of Taking Good Nutrition and Practicing Good Eating Habits - 4 views

  •  
    If our bodies are unhealthy, the physiological process of our body will fail, leading to mental, physical and emotional problems. By eating nutritious food and practicing good eating habits, we can assure of a healthy, comfortable and peaceful life..
Matti Narkia

25-Hydroxyvitamin D3 is an agonistic vitamin D receptor ligand - Lou et al. - J Steroi... - 1 views

  •  
    25-Hydroxyvitamin D(3) is an agonistic vitamin D receptor ligand.
    Lou YR, Molnár F, Peräkylä M, Qiao S, Kalueff AV, St-Arnaud R, Carlberg C, Tuohimaa P.
    J Steroid Biochem Mol Biol. 2009 Nov 26. [Epub ahead of print]
    PMID: 19944755
    doi:10.1016/j.jsbmb.2009.11.011

    In conclusion, 25-hydroxyvitamin D3 is an agonistic vitamin D receptor ligand with gene regulatory and anti-proliferative properties.
Matti Narkia

New topics in vitamin D research - Google Books - 1 views

  •  
    Table of Contents

    Ch. I Is calcidiol an active hormone? 1
    Ch. II Vitamin D as a neurosteroid hormone : from neurobiological effects to behavior 29
    Ch. III Inhibitors of vitamin D hydroxylases : mechanistic tools and therapeutic aspects 67
    Ch. IV Vitamin D analogues as anti-cancer therapies 145
    Ch. V Paricalcitol : a vitamin D2 analog with anticancer effects with low calcemic activity 169
    Ch. VI Vitamin D use among older adults in U.S. : results form national surveys 1997 to 2002 181
    Ch VII Vitamin D deficiency in migrants 199

    Vitamin D is a fat-soluble steroid hormone precursor that contributes to the maintenance of normal levels of calcium and phosphorus in the bloodstream. Strictly speaking, it is not a vitamin since human skin can manufacture it, but it is referred to as one for historical reasons. It is often known as calciferol. The major biologic function of vitamin D is to maintain normal blood levels of calcium and phosphorus. Vitamin D aids in the absorption of calcium, helping to form and maintain strong bones. It promotes bone mineralisation in concert with a number of other vitamins, minerals and hormones. Without vitamin D, bones can become thin, brittle, soft or misshapen. Vitamin D prevents rickets in children and osteomalacia in adults -- skeletal diseases that result in defects that weaken bones. This book gathers international research on the leading-edge of the scientific front.
Matti Narkia

Consuming fructose-sweetened, not glucose-sweetened, beverages increases visceral adipo... - 0 views

  •  
    Consuming fructose-sweetened, not glucose-sweetened, beverages increases visceral adiposity and lipids and decreases insulin sensitivity in overweight/obese humans.
    Stanhope KL, Schwarz JM, Keim NL, Griffen SC, Bremer AA, Graham JL, Hatcher B, Cox CL, Dyachenko A, Zhang W, McGahan JP, Seibert A, Krauss RM, Chiu S, Schaefer EJ, Ai M, Otokozawa S, Nakajima K, Nakano T, Beysen C, Hellerstein MK, Berglund L, Havel PJ.
    J Clin Invest. 2009 May;119(5):1322-34. Epub 2009 Apr 20.
    PMID: 19381015
    doi: 10.1172/JCI37385.

    Studies in animals have documented that, compared with glucose, dietary fructose induces dyslipidemia and insulin resistance. To assess the relative effects of these dietary sugars during sustained consumption in humans, overweight and obese subjects consumed glucose- or fructose-sweetened beverages providing 25% of energy requirements for 10 weeks. Although both groups exhibited similar weight gain during the intervention, visceral adipose volume was significantly increased only in subjects consuming fructose. Fasting plasma triglyceride concentrations increased by approximately 10% during 10 weeks of glucose consumption but not after fructose consumption. In contrast, hepatic de novo lipogenesis (DNL) and the 23-hour postprandial triglyceride AUC were increased specifically during fructose consumption. Similarly, markers of altered lipid metabolism and lipoprotein remodeling, including fasting apoB, LDL, small dense LDL, oxidized LDL, and postprandial concentrations of remnant-like particle-triglyceride and -cholesterol significantly increased during fructose but not glucose consumption. In addition, fasting plasma glucose and insulin levels increased and insulin sensitivity decreased in subjects consuming fructose but not in those consuming glucose. These data suggest that dietary fructose specifically increases DNL, promotes dyslipidemia, decreases insulin sensitivity, and increases visceral adiposity in overweight/obese adults.
Matti Narkia

Diet and Coronary Thrombosis.,Hypothesis and Fact - Yudkin J - Lancet 1957 - Image001.... - 0 views

  •  
    Diet and Coronary Thrombosis., Hypothesis and Fact
    Yudkin J.
    The Lancet 1957.

    (Figure 9 only)
Matti Narkia

Calcium:Magnesium Ratio in Local Groundwater and Incidence of Acute Myocardial Infarcti... - 0 views

  •  
    Calcium:magnesium ratio in local groundwater and incidence of acute myocardial infarction among males in rural Finland.
    Kousa A, Havulinna AS, Moltchanova E, Taskinen O, Nikkarinen M, Eriksson J, Karvonen M.
    Environ Health Perspect. 2006 May;114(5):730-4.
    doi:10.1289/ehp.8438
    PMID: 16675428

    Results of this study with specific Bayesian statistical analysis support earlier findings of a protective role of Mg and low Ca:Mg ratio against coronary heart disease but do not support the earlier hypothesis of a protective role of Ca
Matti Narkia

Retinol-induced Intestinal Tumorigenesis in Min/+ Mice and Importance of Vitamin D Stat... - 0 views

  •  
    "Retinol-induced Intestinal Tumorigenesis in Min/+ Mice and Importance of Vitamin D Status.
    Hetland RB, Alexander J, Berg JP, Svendsen C, Paulsen JE.
    Anticancer Res. 2009 Nov;29(11):4353-60.
    PMID: 20032378

    The effects of life-long dietary exposure, starting in utero, to high retinol, low vitamin D, or high retinol in combination with low vitamin D on intestinal tumorigenesis in Min/+ mice were investigated. In males, high retinol alone significantly increased the number (2.6-fold) and size (1.3-fold) of small intestinal tumours; in females no significant increase in tumour number or size was seen. In both genders, low vitamin D intake alone did not affect intestinal tumorigenesis. In males, intake of the combined high retinol/low vitamin D diet did not further increase the effects caused by high retinol alone. In females, however, the high retinol/low vitamin D-induced increase in tumour number (3.1-fold) and tumour size (1.5-fold) exceeded that of high retinol alone. In conclusion, a high dietary intake of retinol stimulated intestinal tumorigenesis in Min/+ mice. Furthermore, the results indicate a combined effect of high retinol and low vitamin D on tumorigenesis in females"
Matti Narkia

Anticancer Properties of Ganoderma Lucidum Methanol Extracts In Vitro and In Vivo - Nut... - 0 views

  •  
    Anticancer properties of Ganoderma lucidum methanol extracts in vitro and in vivo.
    Harhaji Trajković LM, Mijatović SA, Maksimović-Ivanić DD, Stojanović ID, Momcilović MB, Tufegdzić SJ, Maksimović VM, Marjanović ZS, Stosić-Grujicić SD.
    Nutr Cancer. 2009;61(5):696-707.
    PMID: 19838944
    DOI: 10.1080/01635580902898743

    Anticancer activities of various extracts of the medicinal mushroom, Ganoderma lucidum, have been widely demonstrated and are mainly associated with the presence of different bioactive polysaccharides and triterpenoids. We have evaluated and compared in vitro and in vivo the antitumor effects of two preparations from Ganoderma lucidum: a methanol extract containing total terpenoids (GLme) and a purified methanol extract containing mainly acidic terpenoids (GLpme). Both extracts inhibited tumor growth of B16 mouse melanoma cells inoculated subcutaneously into syngeneic C57BL/6 mice and reduced viability of B16 cells in vitro, whereby GLme exhibited stronger effect. Furthermore, anticancer activity of GLme was demonstrated for the first time against two other rodent tumor cell lines, L929-mouse fibrosarcoma and C6-rat astrocytoma. The mechanism of antitumor activity of GLme comprised inhibition of cell proliferation and induction of caspase-dependent apoptotic cell death mediated by upregulated p53 and inhibited Bcl-2 expression. Moreover, the antitumor effect of the GLme was associated with intensified production of reactive oxygen species, whereas their neutralization by the antioxidant, N-acetyl cysteine, resulted in partial recovery of cell viability. Thus, our results suggest that GLme might be a good candidate for treatment of diverse forms of cancers.
Matti Narkia

Glucose restriction can extend normal cell lifespan and impair precancerous cell growth... - 0 views

  •  
    Glucose restriction can extend normal cell lifespan and impair precancerous cell growth through epigenetic control of hTERT and p16 expression.
    Li Y, Liu L, Tollefsbol TO.
    FASEB J. 2009 Dec 17. [Epub ahead of print]
    PMID: 20019239
    doi: 10.1096/fj.09-149328

    Cancer cells metabolize glucose at elevated rates and have a higher sensitivity to glucose reduction. However, the precise molecular mechanisms leading to different responses to glucose restriction between normal and cancer cells are not fully understood. We analyzed normal WI-38 and immortalized WI-38/S fetal lung fibroblasts and found that glucose restriction resulted in growth inhibition and apoptosis in WI-38/S cells, whereas it induced lifespan extension in WI-38 cells. Moreover, in WI-38/S cells glucose restriction decreased expression of hTERT (human telomerase reverse transcriptase) and increased expression of p16(INK4a). Opposite effects were found in the gene expression of hTERT and p16 in WI-38 cells in response to glucose restriction. The altered gene expression was partly due to glucose restriction-induced DNA methylation changes and chromatin remodeling of the hTERT and p16 promoters in normal and immortalized WI-38 cells. Furthermore, glucose restriction resulted in altered hTERT and p16 expression in response to epigenetic regulators in WI-38 rather than WI-38/S cells, suggesting that energy stress-induced differential epigenetic regulation may lead to different cellular fates in normal and precancerous cells. Collectively, these results provide new insights into the epigenetic mechanisms of a nutrient control strategy that may contribute to cancer therapy as well as antiaging approaches.
Matti Narkia

SUNARC - Sunlight, Nutrition And Health Research Center - 0 views

  •  
    "Cancer mortality rates and multiple sclerosis prevalence rates for U.S. states compared to UVB doses for July"
Matti Narkia

University of Michigan Integrative Medicine - 0 views

  •  
    "University of Michigan Integrative Medicine, an interdisciplinary program, is committed to the thoughtful and compassionate integration of complementary therapies and conventional medicine through the activities of research, education, clinical services and community partnerships. As a healing-oriented approach to medical care, integrative medicine takes into account the whole person (body, mind, spirit and emotion), including all aspects of lifestyle.

    The vision, mission and values of the University of Michigan Integrative Medicine (UMIM) program reflect our belief that patients and our community are best served when all available therapies are considered in concert with an approach that recognizes the intrinsic wholeness of each individual. It also reflects our belief that the best medicine is practiced in collaboration with a wide variety of healthcare professionals and with our patients.

    Our vision: To facilitate healing and wellness of mind, body, heart and spirit through clinical services, research and education.

    Our mission: To provide responsible leadership in the integration of complementary, alternative and conventional medicine.

    Our values: To live and work in balance with the community, the environment and each other. To touch beyond our reach and see beyond our vision."
Matti Narkia

Endothelial-Vasoprotective Effects of High-Density Lipoprotein Are Impaired in Patients... - 0 views

  •  
    Endothelial-Vasoprotective Effects of High-Density Lipoprotein Are Impaired in Patients With Type 2 Diabetes Mellitus but Are Improved After Extended-Release Niacin Therapy.
    Sorrentino SA, Besler C, Rohrer L, Meyer M, Heinrich K, Bahlmann FH, Mueller M, Horváth T, Doerries C, Heinemann M, Flemmer S, Markowski A, Manes C, Bahr MJ, Haller H, von Eckardstein A, Drexler H, Landmesser U.
    Circulation. 2009 Dec 21. [Epub ahead of print]
    PMID: 20026785
    doi: 10.1161/CIRCULATIONAHA.108.836346

    Conclusions-HDL from patients with type 2 diabetes mellitus and metabolic syndrome has substantially impaired endothelial-protective effects compared with HDL from healthy subjects. ER niacin therapy not only increases HDL plasma levels but markedly improves endothelial-protective functions of HDL in these patients, which is potentially more important.
Matti Narkia

NEJM -- Rosuvastatin to Prevent Vascular Events in Men and Women with Elevated C-Reacti... - 0 views

  •  
    Conclusions In this trial of apparently healthy persons without hyperlipidemia but with elevated high-sensitivity C-reactive protein levels, rosuvastatin significantly reduced the incidence of major cardiovascular events. \n\nRosuvastatin to prevent vascular events in men and women with elevated C-reactive protein.\nRidker PM, Danielson E, Fonseca FA, Genest J, Gotto AM Jr, Kastelein JJ, Koenig W, Libby P, Lorenzatti AJ, MacFadyen JG, Nordestgaard BG, Shepherd J, Willerson JT, Glynn RJ; JUPITER Study Group.\nN Engl J Med. 2008 Nov 20;359(21):2195-207. Epub 2008 Nov 9.\nPMID: 18997196
Matti Narkia

Clinical Implications of JUPITER (Justification for the Use of statins in Prevention: a... - 0 views

  •  
    Clinical Implications of JUPITER (Justification for the Use of statins in Prevention: an Intervention Trial Evaluating Rosuvastatin) in a U.S. Population
    Insights From the ARIC (Atherosclerosis Risk in Communities) Study
    Yang EY et al.
    J Am Coll Cardiol, 2009; 54:2388-2395,
    doi:10.1016/j.jacc.2009.10.006

    Conclusions: ARIC participants with elevated hs-CRP and low LDL-C had a CVD event rate of 1.57% per year over 6.9 years, similar to the CVD event rate noted in the JUPITER study placebo group (1.36% per year over 1.9 years). The association of hs-CRP ≥2.0 mg/l with increased CVD risk and mortality regardless of LDL-C provides us a simple method of using age and hs-CRP level for identifying higher risk individuals. (Atherosclerosis Risk in Communities study; NCT00005131)
Matti Narkia

The Vitamin D Research Library is open for your learning pleasure - 0 views

  •  
    "Do Your Vitamin D Research Here
    Links to all the Latest Research Studies

    Welcome to the Vitamin D Research Library.

    Here you'll find links to all of the latest Vitamin D Facts and Research studies, clinical trials and other scholarly Vitamin D Facts. The Research Library is Open 24/7 and is always growing. I'll be adding more and more links all of the time.

    Can't find what you are looking for or just don't want to spend the time doing the research yourself? Chances are, I've already done the Vitamin D Research myself and can answer your question.

    Read Frequently Asked Questions about Vitamin D

    OR

    Ask Your Question About Vitamin D Here

    and I'll answer your Question about Vitamin D Personally based on the best available Vitamin D Research."
1 - 20 of 882 Next › Last »
Showing 20 items per page