great review of data on cardiac damage associated with extreme endurance training. These EEEs, that they are called, are rare in those < 40 and usually involved genetic defects. This article points to aggressive preventive testing in those > 50.
Hashimoto's hypothyroidism is associated with a 44% increase in Heart disease in women under the age o 49. A non-significant change was seen in men. This study found that T4 therapy reduced this risk. A better study would have been if T4 was compared to T4 + T3.
Do Emotions Affect The Structure Of Water?
What you think, feel and speak affect your entire body and change the molecular structure of every drop of water in your body. Make sure that your thoughts, feelings and words are loving and supportive. If they are filled with anger, hate, blame and shame it may lead to toxicity.
The relationship of low testosterone to MetS often is considered to be bidirectional; however, the relationships probably are not direct
Many of the components of the MetS are recognized risk factors for the development of cardiovascular disease (CVD)
Multiple cross-sectional studies have found low TT and low sex hormone binding globulin (SHBG) levels in Caucasian and African-American men with the MetS, irrespective of age
Low TT and SHBG levels also are prevalent in Chinese [7],[8] and Korean [9] men with the MetS
Normally 40%-50% of TT is bound to SHBG, so reducing SHBG levels will decrease TT.
Hyperinsulinism suppresses SHBG synthesis and secretion by the liver
significant increase in SHBG levels occurred after acutely lowering insulin levels in obese men
Estradiol levels are increased in men with the MetS, and they are positively correlated with the number of abnormal components of the MetS.
Although it is known that estrogen will increase SHBG levels, apparently the hyperinsulinism associated with obesity has a greater effect on SHBG levels
Estradiol also can inhibit luteinizing hormone (LH) secretion
Inflammatory cytokines are thought to have a direct effect on the pituitary to reduce LH secretion [15] and also a direct effect on Leydig cell secretion of testosterone
Low TT Levels have been shown to predict development of the MetS in men with normal BMI
Men in the lowest quartiles of serum TT, calculated free testosterone (cFT) and SHBG at baseline had the highest odds ratios for developing the MetS or DM during the 11 years follow-up
More recently, investigators conducting population-based studies have reported that only SHBG is associated with future development of the MetS
Additional evidence that low TT increases the risk of MetS comes from androgen deprivation treatment of prostate cancer
Low TT and low bioavailable testosterone (bT) were each significantly associated with elevated 20 years risk of CVD mortality in an older population in which cause-specific mortality was age, adiposity, and lifestyle-adjusted.
combination of low bT and ATP III-defined MetS is associated with increased cardiovascular mortality in men aged 40 years and above
in elderly men, testosterone may weakly protect against CVD. Alternatively, low TT may indicate poor general health
Muraleedharan and Jones [27] concluded that there is convincing evidence that low T is a biomarker for disease severity and mortality.
The evidence that TRT improves insulin sensitivity and glucose control is conflicted
It is widely recognized that testosterone treatment can reduce fat mass and increase lean body mass; however, until recently most reports have not been associated with much weight loss
Changes in body composition and weight loss are considered potential mechanisms by which testosterone treatment improves insulin sensitivity and glucose control in patients with diabetes. Effects on inflammatory cytokines [38] and changes in oxidative metabolism [39] also have been reported to improve glucose metabolism.
Testosterone replacement therapy has been reported to improve some or all of the components of the MetS.
High fat diet in this small animal study finds an increase in reverse T3 and a decrease in T3. Higher carbohydrate diet provided no significant change.
The starting point for innate immunity activation is the recognition of conserved structures of bacteria, viruses, and fungal
components through pattern-recognition receptors
TLRs are PRRs that recognize microbe-associated molecular patterns
TLRs are transmembrane proteins containing extracellular domains rich in leucine repeat sequences and a cytosolic domain
homologous to the IL1 receptor intracellular domain
The major proinflammatory mediators produced by the TLR4 activation in response to endotoxin (LPS) are TNFα, IL1β and IL6,
which are also elevated in obese and insulin-resistant patients
Obesity,
high-fat diet, diabetes, and NAFLD are associated with higher gut permeability leading to metabolic endotoxemia.
Probiotics,
prebiotics, and antibiotic treatment can reduce LPS absorption
LPS promotes hepatic insulin
resistance, hypertriglyceridemia, hepatic triglyceride accumulation, and secretion of pro-inflammatory cytokines promoting
the progression of fatty liver disease.
In the endothelium, LPS induces the expression of pro-inflammatory, chemotactic, and
adhesion molecules, which promotes atherosclerosis development and progression.
In the adipose tissue, LPS induces adipogenesis,
insulin resistance, macrophage infiltration, oxidative stress, and release of pro-inflammatory cytokines and chemokines.
the gut microbiota has been recently proposed to be an environmental factor involved
in the control of body weight and energy homeostasis by modulating plasma LPS levels
dietary fats alone might not be sufficient to cause overweight and obesity, suggesting that a
bacterially related factor might be responsible for high-fat diet-induced obesity.
This was accompanied in high-fat-fed mice by a change in gut microbiota composition, with reduction in
Bifidobacterium and Eubacterium spp.
n humans, it was also shown that meals with high-fat and high-carbohydrate content (fast-food style western diet) were able
to decrease bifidobacteria levels and increase intestinal permeability and LPS concentrations
it was demonstrated that, more than the fat amount, its composition was a critical modulator of ME (Laugerette et al. 2012). Very recently, Mani et al. (2013) demonstrated that LPS concentration was increased by a meal rich in saturated fatty acids (SFA), while decreased after a
meal rich in n-3 polyunsaturated fatty acids (n-3 PUFA).
this effect seems to be due to the fact that some SFA (e.g., lauric and mystiric acids) are part of the lipid-A component
of LPS and also to n-3 PUFA's role on reducing LPS potency when substituting SFA in lipid-A
these experimental results suggest a pivotal role of CD14-mediated TLR4 activation in the development of
LPS-mediated nutritional changes.
This suggests a link between gut microbiota, western diet, and obesity and indicates that gut microbiota manipulation can
beneficially affect the host's weight and adiposity.
endotoxemia was independently
associated with energy intake but not fat intake in a multivariate analysis
in vitro that endotoxemia activates pro-inflammatory cytokine/chemokine production via NFκB and MAPK signaling in preadipocytes and
decreased peroxisome proliferator-activated receptor γ activity and insulin responsiveness in adipocytes.
T2DM patients have mean values of LPS that are 76% higher than healthy controls
LPS-induced release of glucagon, GH and cortisol, which inhibit glucose uptake, both
peripheral and hepatic
LPSs also seem to induce ROS-mediated apoptosis in pancreatic cells
Recent evidence has been linking ME with dyslipidemia, increased intrahepatic triglycerides, development, and progression
of alcoholic and nonalcoholic fatty liver disease
The hepatocytes, rather than hepatic macrophages, are the cells responsible for its clearance, being ultimately excreted
in bile
All the subclasses of plasma lipoproteins can bind and neutralize the toxic effects of LPS, both in vitro (Eichbaum et al. 1991) and in vivo (Harris et al. 1990), and this phenomenon seems to be dependent on the number of phospholipids in the lipoprotein surface (Levels et al. 2001). LDL seems to be involved in LPS clearance, but this antiatherogenic effect is outweighed by its proatherogenic features
LPS produces hypertriglyceridemia by several mechanisms, depending on LPS concentration. In animal models, low-dose LPS increases
hepatic lipoprotein (such as VLDL) synthesis, whereas high-dose LPS decreases lipoprotein catabolism
When a dose of LPS similar to that observed in ME was infused in humans, a 2.5-fold increase in endothelial lipase was observed,
with consequent reduction in total and HDL. This mechanism may explain low HDL levels in ‘ME’ and other inflammatory conditions
such as obesity and metabolic syndrome
It is known that the high-fat diet and the ‘ME’ increase intrahepatic triglyceride accumulation, thus synergistically contributing
to the development and progression of alcoholic and NAFLD, from the initial stages characterized by intrahepatic triglyceride
accumulation up to chronic inflammation (nonalcoholic steatohepatitis), fibrosis, and cirrhosis
On the other hand, LPS activates Kupffer cells leading to an increased production of ROS and pro-inflammatory cytokines
like TNFα
high-fat diet mice presented with ME, which
positively and significantly correlated with plasminogen activator inhibitor (PAI-1), IL1, TNFα, STAMP2, NADPHox, MCP-1, and
F4/80 (a specific marker of mature macrophages) mRNAs
prebiotic administration reduces intestinal permeability
to LPS in obese mice and is associated with decreased systemic inflammation when compared with controls
Cani et al. also found that high-fat diet mice presented with not only ME but also higher levels of inflammatory markers, oxidative
stress, and macrophage infiltration markers
This suggests that important links between gut microbiota, ME, inflammation, and oxidative stress are implicated in a high-fat
diet situation
high-fat feeding is associated with adipose
tissue macrophage infiltration (F4/80-positive cells) and increased levels of chemokine MCP-1, suggesting a strong link between
ME, proinflammatory status, oxidative stress, and, lately, increased CV risk
LPS has been shown to promote atherosclerosis
markers of systemic inflammation such as circulating bacterial endotoxin
were elevated in patients with chronic infections and were strong predictors of increased atherosclerotic risk
As a TLR4 ligand, LPS has been suggested to induce atherosclerosis development and progression, via a TLR4-mediated inflammatory
state.
The prevalence of hypogonadism (often defined as serum testosterone < 300 ng dl−1 ) ranges from 6% [10] to as high as 38%
The process of BPH, however, continues as men age and despite the fact their serum testosterone decreases
Liu et al. [12] demonstrated that in a group of older males (mean age 59.8 years) that there was not a significant correlation of serum testosterone levels (total, free or bioavailable) with either prostate volume or International Prostate Symptom Score (IPSS)
in eugonadal men, studies have demonstrated that the prostate can increase in volume by approximately 12%
There seems to be little doubt that the treatment with testosterone of a young hypogonadal male leads to significant growth of the prostate
Behre et al. [22] demonstrated increased prostate volume and prostate-specific antigen (PSA) levels in hypogonadal men
Most studies, however, have shown no effect of exogenous androgens on PSA or prostate volume for older hypogonadal males
saturation model
They argue that the prostate is relatively insensitive to changes in androgen concentration at normal levels or in mild hypogonadism because the AR is saturated by androgens and therefore maximal androgen-AR binding is achieved. Conversely, the prostate is very sensitive to changes in androgen levels when testosterone is low
visceral obesity (one of the most significant components of metabolic syndrome) is associated with prostate volume and influences prostate growth during TRT.
This hypothesis of inflammation induced LUTS is also argued to be a mechanism for improvement of LUTS with PDE5I
The concept, therefore, that treatment with TRT of hypogonadal males with metabolic syndrome might lead to improvement/stabilization of their LUTS, appears to be confirmed in recent work by Francomano et al.
There was also an improvement in components of the patient's metabolic syndrome (such as BMI, waist circumference, hemoglobin A1c [HbA1c], insulin sensitivity, and lipid profile) as well as inflammatory markers and C-reactive protein.
They concluded that TRT was safe in this group of men, and hypothesize that TRT mitigates the pro-inflammatory factors associated with metabolic syndrome.
Authors review the literature behind Testosterone and BPH. The authors highlight the 4 proposed theories behind BPH: Testosterone, Estrogen, inflammation, and metabolic.
The conclusion is mixed: pointing out that no high level of evidence exists on either side of the debate of Testosterone and BPH.
Total serum testosterone consists of free testosterone (2%–3%), testosterone bound to sex hormone binding globulin (SHBG) (45%) and testosterone bound to other proteins (mainly albumin −50%)
Testosterone binds only loosely to albumin and so this testosterone as well as free testosterone is available to tissues and is termed bioavailable testosterone
Testosterone bound to SHBG is tightly bound and is biologically inactive
Bioavailable and free testosterone are known to correlate better than total testosterone with clinical sequelae of androgenization such as bone mineral density and muscle strength
peak levels seen in the morning following sleep, which can be maintained into the seventh decade
Samples should always be taken in the morning before 11 am
The reliable measurement of serum free testosterone requires equilibrium dialysis. This is not appropriate for clinical use as it is very time consuming and therefore expensive.
With increasing age, a greater number of men have total testosterone levels just below the normal range or in the low-normal range. In these patients total testosterone can be an unreliable indicator of hypogonadal status.
It is advised that at least two serum testosterone measurements, taken before 11 am on different mornings, are necessary to confirm the diagnosis.
Patients with serum total testosterone consistently below 8 nmol/l invariably demonstrate the clinical syndrome of hypogonadism and are likely to benefit from treatment. Patients with serum total testosterone in the range 8–12 nmol/l often have symptoms attributable to hypogonadism and it may be decided to offer either a clinical trial of testosterone treatment or to make further efforts to define serum bioavailable or free testosterone and then reconsider treatment. Patients with serum total testosterone persistently above 12 nmol/l do not have hypogonadism and symptoms are likely to be due to other disease states or ageing per se so testosterone treatment is not indicated.
Total testosterone levels fall at an average of 1.6% per year whilst free and bioavailable levels fall by 2%–3% per year.
With advancing age there is also a reduction in androgen receptor concentration in some target tissues and this may contribute to the clinical syndrome of LOH
Metabolic clearance declines with age
Gonadotrophin levels rise during aging (Feldman et al 2002) and testicular secretory responses to recombinant human chorionic gonadotrophin (hCG) are reduced
There are changes in the lutenising hormone (LH) production which consist of decreased LH pulse frequency and amplitude, (Veldhuis et al 1992; Pincus et al 1997) although pituitary production of LH in response to pharmacological stimulation with exogenous GnRH analogues is preserved
the decreases in testosterone levels with aging seem to reflect changes at all levels of the hypothalamic-pituitary-testicular axis
Heavy Duty Wheelchair specialy design for disabled and handicapped persons. They two types of Heavy Duty Wheelchair for handicapped and disability product like-
Karma 8020 X Heavy Duty Wheelchair
Karma 8520 Heavy Duty Wheelchair
Karma 8020 X Heavy Duty Wheelchair
Karma 8020 or Karma KM-8020 Heavy Duty Wheelchair specialy design for disabled and handicapped persons. Some features are mentioned below-
Features
It comes with detachable swing away footrests.
The wheel chair has flip-back armrests.
It comes with centre of gravity adjustment.
The wheel chair has wide profile casters.
Seat Size 20'' inch & 22'' inch
Total Weight 17 K.G.
Karma 8020 X Heavy Duty Wheelchair Item Code and Price
Item Code: WCI-52
MRP: Rs 45162
Our Price: Rs 32900
Net Price: Rs 32242( Apply Coupon Code 'MSW2365' Get 2% Discount )
Karma 8520 Heavy Duty Wheelchair
Karma 8520 or Karma KM-8520 Heavy Duty Wheelchair specialy design for disabled and handicapped persons. Some features are mentioned below-
Features
It comes with detachable swing away footrests.
The wheel chair has flip-back armrests.
It comes with centre of gravity adjustment.
The wheel chair has wide profile casters.
Seat Size 20'' inch & 22'' inch
Total Weight 17 K.G.
Karma 8520 Heavy Duty Wheelchair Item Code and Price
Item Code: WCI-29
MRP: Rs 32085
Our Price: Rs 24063
Net Price: Rs 23582( Apply Coupon Code 'MSW2365' Get 2% Discount )
Heavy Duty Wheelchair Online Purchases:
This Website is only a venue where Users may meet and interact with us for their sale and purchase transactions.
The commercial / contractual terms include without limitation price, shipping costs, date, period, mode of delivery, warranties related to products and services including after sales services related to product(s) and services, etc., over which we have complete control and same may be the subject to change.
The agreement between you and wheelchairindia.com is subject to the following terms and cond
Heavy Duty Wheelchair specialy design for disabled and handicapped persons. They two types of Heavy Duty Wheelchair for handicapped and disability product like-
Karma 8020 X Heavy Duty Wheelchair
Karma 8520 Heavy Duty Wheelchair
Karma 8020 X Heavy Duty Wheelchair
Karma 8020 or Karma KM-8020 Heavy Duty Wheelchair specialy design for disabled and handicapped persons. Some features are mentioned below-
Features
It comes with detachable swing away footrests.
The wheel chair has flip-back armrests.
It comes with centre of gravity adjustment.
The wheel chair has wide profile casters.
Seat Size 20'' inch & 22'' inch
Total Weight 17 K.G.
Karma 8020 X Heavy Duty Wheelchair Item Code and Price
Item Code: WCI-52
MRP: Rs 45162
Our Price: Rs 32900
Net Price: Rs 32242( Apply Coupon Code 'MSW2365' Get 2% Discount )
Karma 8520 Heavy Duty Wheelchair
Karma 8520 or Karma KM-8520 Heavy Duty Wheelchair specialy design for disabled and handicapped persons. Some features are mentioned below-
Features
It comes with detachable swing away footrests.
The wheel chair has flip-back armrests.
It comes with centre of gravity adjustment.
The wheel chair has wide profile casters.
Seat Size 20'' inch & 22'' inch
Total Weight 17 K.G.
Karma 8520 Heavy Duty Wheelchair Item Code and Price
Item Code: WCI-29
MRP: Rs 32085
Our Price: Rs 24063
Net Price: Rs 23582( Apply Coupon Code 'MSW2365' Get 2% Discount )
Heavy Duty Wheelchair Online Purchases:
This Website is only a venue where Users may meet and interact with us for their sale and purchase transactions.
The commercial / contractual terms include without limitation price, shipping costs, date, period, mode of delivery, warranties related to products and services including after sales services related to product(s) and services, etc., over which we have complete control and same may be the subject to change.
The agreement between you and wheelchairindia.com is subject to the following terms and cond
Ergonomic wheelchair series provides users with a large selection of ultra lightweight wheelchairs that can help improve life. This series has features that include a high strength lightweight frame, foldable and easy to store, breathable anti-bacterial, anti-staining, removable and machine washable cushion. Also includes the exclusive S-Shape Seating System, which provides increased stability, better weight distribution and lowers the risk of pressure sores and spinal injury. The patented S-Shape Seating System that comes with every wheelchair model in this series provides an ergonomic seating frame that conforms and flexes to the shape of body.
Karma S Ergo 115 Wheelchair:
This model features our S-Shape Seating System and is our number one best seller for many reasons. At a mere 11.3 kg in weight with detachable foot rest and many features such as removable machine washable and dry-able cushions treated by AEIGIS treated anti-microbial coated seating system.
Karma S-Ergo 115 Wheelchair Features:
Ergonomic Handrims & S-Shape Ergonomic Seating System
Fixed armrest w/ wider concave armpads
Swing In & Away Footrests
Backrest Pouch attached to the upholstery
24" flat free polyurethane tires, high tread, flat free wheels
Seat width: 16"x17" or 18"x17" or 20"x 17"
Silver 1/4" Aegis Anti-Bacterial Upholstery, washable
Folding backrest / folding seat for easy traveling
"Tube-in Center" foot-plate, assures better side leg support
High strength, starting weight at only 11.3 kg. (w/o footrests)
7×1" Polyurethane front casters
Upholstery: Black breathable mesh bottom & top AEIGIS
Frame Color: Pearl Silver or Rose Red
Weight Capacity of 115 kg.
Karma S-Ergo 115 Wheelchair Measurements:
Seat Width 16 inch., 18 inch., 20 inch.
Seat Depth 17 inch.
Armrest Height 8 inch.
Seat Height 19 inch.
Back Height 17 inch.
Overall Height 36 inch.
Overall Open Width 23 inch., 25 inch., 27 inch.
Folded Width 12 inch.
Overall Length 39 i
study proposes that arachidonic acid and DHA improve cognition in individuals with age related cognition changes and organic brain damage (Alzheimer's, brain lesions...)
Lightweight Ergonomic Wheelchair is designed to cradle patients in comfort while reducing risk of pressure sores, relieving pressure, dispersing weight evenly and improving stability. The Ergo Lite Wheelchair is easy to lift and its small size makes it ideal for travel. Weighing only 19.8 lbs., the chair is easy to store and transport. The chair folds down the middle like a standard wheelchair and the backrest also folds down to make the wheelchair even more compact. Also features companion brakes located on handles for added safety and comfort. Ergonomic Wheelchairs are ultra lightweight and have a supportive seating system to provide pressure relief and anti-slippage giving the user excellent stability and posture support. .
Better Ergonomic wheelchair for Comfort: Sit for any length of time in a fixed position in a traditional transport chair and it will feel how painful bad ergonomics can be. It is fully reclined 25° angle, which minimizes slumping and sliding. And because of this can be easily adjusted with the push of a button, it can change positions frequently over time, reducing the chance of secondary conditions that may include pressure sores.
Designed to Fit Almost Everyone: It is one of the most extensively researched seat profiles specifically design to accommodate users ranging in size from small women to large men, with the greatest amount of comfort and proper posture/spinal support.
The Karman wheelchairs are exceptional in both their function and style. With comfort built right into the frame of a wheelchair eliminating the need for a "thick cushion" to add comfort, S-Shape seat allows the wheelchair to be "ergonomically correct" putting into a seating position and comfort conforming to natural body's curves. These chairs are built for comfort and quality to stand the test of time.
Ergonomic Wheelchair is Right: Ergonomic Wheelchair is designed for users who will spend a large amount of time in the chair. This wheelchair'
Pristine Flex Ostrich Mobility Wheelchair
Pristine is the most stylish powered wheelchair with revolutionary Split Frame Chassis (SFC). This indoor outdoor mobility equipment gives great comfort, stability and safety even in the toughest outdoor conditions. The body panels and the shopping basket make this equipment best suited for your kind of lifestyle.
Pristine Flex Ostrich Mobility Wheelchair Features
Split Frame Chassis (SFC).
Call alarm, fault alarm, reverse alarm (on request).
Low voltage alarm.
Key pad locking.
Mobility cut-off while charging.
Auto shut-off after 3 minutes.
Five speed selector.
Length adjustable joystick control unit (can be changed from left to right and vice versa).
Foldable, height adjustable and angle adjustable foot rest.
Wide arm rest with height and width adjustment.
Bucket seat with headrest and lap belt.
Seat can be folded for transportation.
Seat reclines 25 degrees in 6 steps.
Removable and width adjustable calf support.
Reflectors as per standards.
Head lamp.
Tail lamp.
Pristine Flex Ostrich Mobility Wheelchair Specifications
Load capacity: 110 Kg
Speed: 9.5 Kmph max
Speed selection: 5 speeds, Speed 1- 1.5Kmph, Speed 2-2.5 Kmph, speed 3- 4.8 Kmph, Speed 4- 7.2 Kmph, Speed 5- 9.5 Kmph
Power: 320W
Motor speed: 5300 rpm
Gear ratio: 32:1
Brake: Electromagnetic
Permissible Gradeability: 12 Degrees
Drive range: 32-35 Km
Ground clearance:2.5 inches
Turn circle radius: 620mm
Tire: Puncture free foam filled rubber tires
Front- Tire diameter: 220X55 mm, Rear- Tire diameter: 320X72mm, Anti tippers-1 inch solid
Battery: 24 Volt 48 Ah Sealed Maintenance Free VRLA Charger Input-230/240 Volts AC Single phase, Output-24 Volts-4 Amps DC
Overall length with footrest (at 90 degree):1180mm, Overall width:640mm, Overall height: 1250mm, Overall height after folding the seat: 690mm, Overall weight: 102 Kg
Seat depth: 500mm, Seat width:480mm, Backrest height: 540mm (without head rest), Backrest width: 440mm, Se
Disabled people not only have these challenges to face, but a number of other concerns. Disabled people who are blind, deaf and are wheelchair bound face additional challenges. Issues such as accessibility, being able to understand words that are spoken or written can make travel doubly difficult. Ultralight manual wheelchair are essential for ensuring a user's ability to use their wheelchair in the most functional, efficient, and healthy way possible. For individuals who have utilized an ultralight manual wheelchair for a number of years, it is common for them to know exactly how their wheelchair should be set-up from the precise seat to floor height measurement and vertical position of the rear wheel. On the other hand, a newer user may require adjustability to meet their changing needs over time as they continues to recover and/or adapt to using a wheelchair.
Karma KM 2500 L Big Wheel Wheelchair:
Karma KM 2500 L Wheelchair Specifications:
Width 18"
Front/Rear Wheels 6" to 22"
Seat Width 47cm
Seat Depth 40cm
Overall Width 66cm
Overall Collapsed Width 36cm
Armrest Height 21cm
Overall Length 90cm
Seat Height 47cm
Backrest Height 38cm
Overall Height 86cm
Weight 11.kg
Karma KM 2500 L Wheelchair Seat and Back:
AEGIS Microbe Shield Approved by the FDA, EPA, EU, etc., bonded anti-microbial barrier upholstery protects from odor, staining and deterioration from bacteria, fungus and other microorganisms. It is a shield for your health.
Karma KM 2500 L Wheelchair Extended Armrest
By simulating the natural position of arms, the extended armrest design is ergonomic and creates bigger seating space.
An Ultra lightweight wheelchair (11 kg) with a compact design for either attendant assisted or self propelling users.
The use of aircraft-grade aluminium alloy and double cross brace provide this model with outstanding strength and durability.
Karma Healthcare KM-2500 L Premium Wheelchair is amazingly light and compact transit wheelcha
Lightweight Wheelchair is loaded with features and adjust ability. Almost everything can be customized to fit needs, from a height and position adjustable back to an adjusting rear axle. The semi-pneumatic spoke wheels not only look stylish, but provide a smooth and true roll, giving more control over where are going. This Ultra Lightweight Wheelchairs is available in two seat widths and with either swing away foot rests or elevating foot rests to fit needs. Ultralight manual wheelchair are essential for ensuring a user's ability to use their wheelchair in the most functional, efficient, and healthy way possible. For individuals who have utilized an ultralight manual wheelchair for a number of years, it is common for them to know exactly how their wheelchair should be set-up from the precise seat to floor height measurement and vertical position of the rear wheel. On the other hand, a newer user may require adjust ability to meet their changing needs over time as they continues to recover and/or adapt to using a wheelchair.
Karma KM 2500 Small Wheel Wheelchair:
Karma KM 2500 Small Wheel Wheelchair Specifications:
Width 18"
Front/Rear Wheels 6" to 14"
Seat Width 47cm
Seat Depth 40cm
Overall Width 66cm
Overall Collapsed Width 36cm
Armrest Height 21cm
Overall Length 90cm
Seat Height 47cm
Backrest Height 38cm
Overall Height 86cm
Weight 9.2 k.g.
Karma KM 2500 Small Wheel Wheelchair Seat and Back:
AEGIS Microbe Shield Approved by the FDA, EPA, EU, etc., bonded anti-microbial barrier upholstery protects from odor, staining and deterioration from bacteria, fungus and other microorganisms. It is a shield for your health.
Karma KM 2500 Small Wheel Wheelchair Extended Armrest:
By simulating the natural position of arms, the extended armrest design is ergonomic and creates bigger seating space.
An Ultra lightweight wheelchair (9.2 kg) with a compact design for either attendant assisted or self propelling users.
The use of aircr
If you are looking for a cosmetic change and you don't have sufficient funds in your savings. In these situations you can best make use of the provision of plastic surgery loans and get the quick cash support from reliable lender. With these loans, you can easily afford your cosmetic surgery expenses in hassle free manner.