In this study, flu vaccine is found to reduce MI risk by 45%. Now, the take home point here is that those with cardiovascular compromise need to avoid the flu--not, that the flu vaccine by itself reduces the risk. The authors of this study fail to state the low success of the flu vaccine in the prevention of the flu. They also fail to state the increased inflammation as a result of the vaccine itself. A better approach would be to use natural therapies i.e.. vitamin D that have been shown to prevent the flu.
Study finds increased risk of sudden MI in men with low Testosterone and elevated Estradiol. In converse, increased risk is associated with elevated Estradiol in women.
Thyroid hormone plays significant role in cardiac remodeling after acute myocardial infarction. Thyroid hormone, particularly T3 as the vast majority of T3 is produced in heart tissue via D1 enzymatic activity, improves cardiac contractility, reduces systemic vascular resistance, reduces cardiac work load, decreases blood pressure, improves cardiac metabolism, and thus improves outcomes post MI.
Significantly higher estradiol levels found in men with a history of MI. These men would likely have an increase in adverse effects associated with Testosterone therapy.
Mi GFR es 20,y tengo la enfermedad renal diabética ,cómo puado aumentar mi GFR? Para una persona con enfermedad renal diabética y GFR 20, no hay cura, pero los tratamientos naturales pueden ayudar a aumentar la función renal, así como para detener la progresión de la enfermedad.
Retrospective study finds no increase in new MI and/or stroke in men with Testosterone therapy. There are some men that Testosterone therapy is cardioprotective and some that are not.
overall mortality and CV mortality were inversely associated with serum T concentrations.
men with low serum T, defined as < 8.7 nmol l−1 (250 ng dl−1 ), demonstrated significantly greater all-cause mortality than men with higher serum T (hazard ratio [HR]: 2.24; 95% CI: 1.41-3.57), as well as greater CV mortality
lower T levels were significantly associated with the presence of any CV disease
more than 30 years of studies suggesting that low levels of T represent an increased risk for CV and overall mortality,
lower serum T concentrations also are associated with CV disease, including incident coronary artery disease [17],[18],[19] and atherosclerosis,
the actual rate of adverse events was only half as great in the T group (123 events in 1223 men at risk = 10.1%) as in the untreated group (1587 events in 7486 men = 21.2%)
The study by Vigen et al. [7] has already undergone two published corrections,
29 medical societies have called for retraction of the article, asserting "gross data mismanagement and contamination," that rendered the study "no longer credible
Mortality in T-treated men was reduced by approximately half in treated men compared with untreated men, at 10.3% versus 20.7%, respectively
The mortality rate for men who received TTh was 3.4 deaths per 100 person-years, and 5.7 deaths per 100 person-years in untreated men
HR of 0.61 (95%CI: 0.42-0.88; P = 0.008), indicating a significant reduction in mortality with TTh
men in the highest prognostic MI risk quartile, treatment with TTh was associated with reduced risk
tripling in T prescriptions in the US over the last decade
a majority of observational studies have found that low endogenous serum T levels are associated with increased mortality.
Men who received TTh were able to exercise significantly longer without ischemia compared with men who received placebo
In men with congestive heart failure, those who received T demonstrated greater walking distance and other functional endpoints compared with those who received placebo
TTh has been shown uniformly and repeatedly to improve several known CV risk factors, including reduced fat mass, body fat percent, and waist circumference, and increased lean mass
improved glycemic control
reductions in insulin resistance.
the evidence strongly points to improved CV status with normal serum T or treatment with TTh in men with TD
analysis of health insurance claims data that reported a 36% increased rate of nonfatal MI in the 90d following receipt of a T prescription compared with the 12 prior months.
Comparison with men who received a prescription for a phosphodiesterase type 5 inhibitor (PDE5i) revealed no increased rate of MI following the prescription
Great review by Morgentaler of Testosterone and CVD. He highlights the significant flaws in the JAMA and the NEJM articles of Testosterone therapy risks. Morgentaler highlights the significant evidence that points to low T and increased risk of CVD.
On contention I have, is Morgantaler seems to flip aside the massive uptick of Testosterone use in the US as compared to other countries. The evidence definitely points to Testosterone therapy as being safe in those with low T, but there is definitely a problem of significant Testosterone doping that is taking place as well.
Soy un paciente del nefritis lúpica y recientemente mi nivel de creatinina aumentó a 5.5, excepto que no tengo síntomas mucho. Creo que sólo necesito hacer unos pequeños cambios en mi dieta, pero mi doctor dijo que tengo que tomar diálisis. Quiero saber es la diálisis es una elección obligada para mí?
Only abstract available here, but EDTA chelation found to reduce risk of death, reinfarction, stroke, hospitalization, angina in individuals with diabetes and prior MI from 38% to 25%.
vitamin E, in doses up to 800 IU daily, found to be safe and effective in reducing risk of non-fatal MI in individuals with symptomatic coronary atherosclerosis.
Hola, mi hijo tiene 6 años de edad, refractario a tratamiento de cordicoides, tiene síndrome nefrótico y esta con una creatinina de 6.6. ¿Es muy riesgos de ese valor? "Hola, mi hijo tiene 6 años de edad, refractario a tratamiento de cordicoides, tiene síndrome nefrótico y esta con una creatinina de 6.6.
Estoy un sobreviviente de cáncer fue tratado con la medicina y la radiación hace 2 años mi nivel BUN es 28 y proteína creatinina 7.5 tiene que entrar en pronto alguna idea de por qué mi bollo es tan alta de repente
Soy un paciente de insuficiencia renal ,y ser sufrido por la enfermedad renal 10 años ,ahora mi función renal sólo hay 23,mi calidad de vida es muy baja .cada día estoy muy desesperado ,¿Cómo puedo mejorar la función renal ,puedes ayudarme ?Muchas gracias .
Soy un paciente de la nefropatía diabética ,ahora mi creatinina es 300,y mi calidad de la viada es muy baja ,pues necesito tomar la diálisis para controlarla ? En general, después de que el paciente se cayó en la enfermedad renal en etapa terminal, la diálisis será en la demanda.Pero si el paciente tiene mucho síntomas ,puede tomar la diálisis por adelantado.
Qué debo hacer mi nivel de creatinina es 1.8 pero GFR es 26? Cuál es el problema?Sabes,el nivel de creatinina y GFR son dos de los indicadores más comunes de enfermedad renal, pero relativamente GFR es más sensible que el nivel de creatinina.En tu caso, aunque tu nivel de creatinina no es muy alto, solo 1,8, pero tu GFR es de 26 que indican su enfermedad ahora ha progresado en la etapa 4 de la enfermedad renal.
Study finds strong correlation between Apo B and Apo B:apo A1 in fatal MI risk in men and women. Apo B was a stronger than LDL in predicting risk in men and women. Apo A1 was noted to be protective, but the stats were less strong.