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Effects of chronic estradiol treatment on the thyr... [BMC Res Notes. 2009] - PubMed re... - 0 views

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    too little/ too much estrogen can negatively effect thyroid function; hormone balance one of the 5 points to health
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Small Differences in Thyroid Function May Be Important for Body Mass Index and the Occu... - 0 views

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    TSH doesn't have to be above normal for your weight to be negatively affected
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Vitamin D3 Is More Potent Than Vitamin D2 in Human... [J Clin Endocrinol Metab. 2010] -... - 0 views

  • Given its greater potency and lower cost, D3 should be the preferred treatment option when correcting vitamin D deficiency.
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    Vitamin D3 better absorbed than D2: and should be the preferential treatment in vitamin D deficiency
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C-Reactive Protein as a Cardiovascular Risk Factor : More Than an Epiphenomenon? -- Lag... - 0 views

  • CRP constitutes an independent cardiovascular risk factor
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    CRP is an independent marker for future cardiovascular disease
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Kidney International - Abstract of article: Inflammation enhances cardiovascular risk a... - 0 views

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    Inflammation (point 5 of 5 points of wellness) increases CRP, which increases cardiovascular risk
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Inflammation as a Cardiovascular Risk Factor -- Willerson and Ridker 109 (21 Supplement... - 0 views

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    statins do lower CRP reducing inflammation: one of primary benefits of statin therapy
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Prospective Study of C-Reactive Protein and the Risk of Future Cardiovascular Events Am... - 0 views

  • CRP is a strong independent risk factor for cardiovascular disease that adds to the predictive value of risk models based on usual factors alone.
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    CRP is marker for cardiovascular risk in women
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Inflammatory bio-markers and cardiovascular risk prediction - Blake - 2002 - Journal of... - 0 views

  • Inflammatory processes are now recognized to play a central role in the pathogenesis of atherosclerosis and its complications. Plasma levels of several markers of inflammation have been found to be associated with future cardiovascular risk in a variety of clinical settings.
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    inflammation (point 5 of the 5 points of wellness) shown to play central role in atherosclerosis.  See CRP
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Interrelationships Among Circulating Interleukin-6, C-Reactive Protein, and Traditional... - 0 views

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    IL-6 and CRP reveal systemic vascular inflammation
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Neuroleptic medications inhibit complex I of the e... [Ann Neurol. 1993] - PubMed result - 0 views

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    neuroleptic drugs increase risk of Parkinson's disease by 20-40%
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Coenzyme Q10 in neuromuscular and neurodegenerativ... [Curr Drug Targets. 2010] - PubMe... - 0 views

  • a deficit in CoQ10 status has been determined in a number of neuromuscular and neurodegenerative disorders
  • A secondary loss of CoQ10 status following HMG-Coa reductase inhibitor (statins) treatment has be implicated in the pathophysiology of the myotoxicity associated with this pharmacotherapy
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    CoQ10 contributes to neurodegenerative disease, Parkinson's, Alzheimer's, ALS..., in some
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Coenzyme Q10 in neurodegenerative diseases. [Curr Med Chem. 2003] - PubMed result - 0 views

  • supplemental coenzyme Q(10) can slow the functional decline in these disorders, particularly Parkinson's disease.
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    CoQ10 may slow some aspects of Parkinson's disease
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Coenzyme Q treatment of neurodegenerative diseases... [Mitochondrion. 2007] - PubMed re... - 0 views

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    CoQ10 shows promise in neurodegenerative disorders
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Oxidative stress, mitochondrial dysfunction and ce... [J Neurol Sci. 2005] - PubMed result - 0 views

  • There is significant evidence that the pathogenesis of several neurodegenerative diseases, including Parkinson's disease, Alzheimer's disease, Friedreich's ataxia (FRDA), multiple sclerosis and amyotrophic lateral sclerosis, may involve the generation of reactive oxygen species (ROS) and/or reactive nitrogen species (RNS) associated with mitochondrial dysfunction
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    Mitochondrial dysfunction at heart of diseases such as Parkinson's, Alzheimers, MS, ALS..
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Coenzyme Q10 administration and its potential for ... [Biofactors. 1999] - PubMed result - 0 views

  • hese studies therefore raise the prospect that administration of CoQ10 may be useful for the treatment of neurodegenerative diseases.
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    CoQ10 may be used to slow progression of neurodegenerative diseases such as Alzheimers, Parkinson's, ALS...
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Coenzyme Q10 as a possible treatment for neurodege... [Free Radic Res. 2002] - PubMed r... - 0 views

  • CoQ10 can significantly extend survival, delay motor deficits and delay weight loss and attenuate the development of striatal atrophy in a transgenic mouse model of HD
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    CoQ10 shown to shown to slow progression of the neurodegenerative disease: Huntington's disease
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Coenzyme Q10 effects in neurodegenerative disease. [Neuropsychiatr Dis Treat. 2009] - P... - 0 views

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    CoQ10 shown to be beneficial in treatment of neurodegenerative diseases, Parkinson's, Alzheimer's, ALS, Huntington's, in animal models
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The evidence basis for coenzyme Q therapy in oxida... [Mitochondrion. 2007] - PubMed re... - 0 views

  • In some mitochondrial diseases and other commoner disorders such as cardiac disease and Parkinson's disease low mitochondrial or tissue levels of CoQ10 have been demonstrated providing an obvious rationale for supplementation
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    Coq10 in Parkinson's disease and cardiac disease beneficial
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Coenzyme Q10: a review of its promise as a neuropr... [CNS Spectr. 2007] - PubMed result - 0 views

  • Experimental studies in animal models suggest that CoQ10 may protect against neuronal damage that is produced by ischemia, atherosclerosis and toxic injury.
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    CoQ10 and neuroprotection
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Friedreich's Ataxia: disease mechanisms, antioxida... [Biofactors. 2003] - PubMed result - 0 views

  • This therapy clearly showed a rapid and sustained increase in the energy generated by the FRDA heart muscle, nearly returning to normal levels
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    CoQ10 and Vitamin E shown to improve mitochondrial function in clients with Friedreich's Ataxia
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