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Scientists may debate the accuracy of this equivalency, but most people agree that humans live many times longer than their furry friends. And, as it happens, these friends have features that uniquely qualify them to help us speed biomedical research.

The complex systems of high school dating and chemical reactions may have more in common than you think. Explore five rules for speeding up chemical reactions in the lab that might just land you a date to a dance!

In 1977, a University of Oxford statistician named Richard Peto pointed out a simple yet puzzling biological fact: We humans should have a lot more cancer than mice, but we don't. Dr. Peto's argument was beguilingly simple. Every time a cell divides, there's a small chance it will gain a mutation that speeds its growth. Cells that accumulate several of these mutations may become cancerous. The bigger an animal is, the more cells it has, and the longer an animal lives, the more times its cells divide. We humans undergo about 10,000 times as many cell divisions as mice - and thus should be far more likely to get cancer.

Horn removal through gene editing is a win-win both for the animal and for the farmer, says Muir, who did not take part in this research. "These findings show that you can take highly desirable genes from animals and move them to other members of their species," he told Popular Science. "One could achieve the same results with natural breeding, but gene editing greatly speeds up the process, reducing the time it takes to accomplish the goal from centuries to years."

Around the world, disease-carrying mosquitoes are advancing at speed, taking viruses such as dengue and Zika, plus a host of lesser-known ills such as chikungunya and St. Louis encephalitis, into new territories from Europe to the Pacific. "The concern is that we have these species spreading everywhere. Today the focus is on Zika but they can carry many different viruses and pathogens," said Anna-Bella Failloux, head of the department that tracks mosquito viruses at France's Institut Pasteur.

This directed case study examines differences between the exponential and logistic growth models in biology and how they are applied to solve real life problems. The narrative follows a student returning to the United States as he tries to assess his possible exposure to Middle East Respiratory Syndrome (MERS). To better understand his risk, James needs to get up to speed on a variety of topics including the difference between infection, transmission, virulence, etc., and how these topics can be mathematically modeled not only in relation to MERS, but also with respect to Ebola and influenza. This case was designed for use in the second semester of a biocalculus course or a course involving ordinary differential equations, which are appropriate for second year undergraduate students majoring in biology, pre-med, and bio-mathematics. These students typically have completed a semester of calculus and one year of general biology. The case provides an opportunity for students to develop their understanding of differential equations and increase their appreciation of mathematics as it applies to solving a problem of biology.

Your task is to compare two groups of mice to see how well they have learned the platform location. Two reliable measures of learning in mice are 1) the speed at which they find the platform, and 2) the route which they take to the platform (i.e. do they go straight to the platform or do they get a little lost along the way?).  

Within every cell in our body, two copies of a tumor suppressor gene called BRCA1 are tasked with regulating the speed at which cells divide. Michael Windelspecht explains how these genes can sometimes mutate, making those cells less specialized and more likely to develop into cancer.

The Rare Disease Foundation is focussed on linking basic science and clinical practice to increase the efficiency of rare disease research. This model is called Translational Care. This model drives patient based, treatment focussed research projects from disease characterization to treatment with greater efficiency. By incorporating research, astute clinician observation and parental knowledge into the various stages of rare disease research we impact the speed of discovery and the way rare conditions are managed.

Every second of your life you are under attack. Bacteria, viruses, spores and more living stuff wants to enter your body and use its resources for itself. The immune system is a powerful army of cells that fights like a T-Rex on speed and sacrifices itself for your survival. Without it you would die in no time. This sounds simple but the reality is complex, beautiful and just awesome. An animation of the immune system.

Most colon cancers may be caused by infections with bacteria that are normally found in cows. For decades we have known that Streptococcus gallolyticus gallolyticus (SGG) is sometimes found in colon tumours, but now the microbes have been found to directly cause tumour growth in mice.

Within every cell in our body, two copies of a tumor suppressor gene called BRCA1 are tasked with regulating the speed at which cells divide. Michael Windelspecht explains how these genes can sometimes mutate, making those cells less specialized and more likely to develop into cancer.

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