Vitamin D: the alternative hypothesis.
Albert PJ, Proal AD, Marshall TG.
Autoimmun Rev. 2009 Jul;8(8):639-44. Epub 2009 Feb 12. Review.
PMID: 19393200
Emerging molecular evidence suggests that symptomatic improvements among those administered vitamin D is the result of 25-D's ability to temper bacterial-induced inflammation by slowing VDR activity. While this results in short-term palliation, persistent pathogens that may influence disease progression, proliferate over the long-term.