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Tero Toivanen

Patrick Ecker: AAC - 2 views

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    This site is intended to be an active community for sharing graphics, pictures, and visual supports for children with disabilities. Parents, professionals, and others can share their ideas and help children learn to use language and communicate the best they can.
Tero Toivanen

NDAR - National Database for Autism Research Home Page - 2 views

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    NDAR is a secure bioinformatics platform for scientific collaboration and data-sharing for Autism Spectrum Disorder (ASD) investigators.
Amanda Kenuam

Simple Storytelling for Special Education Students with Storybird - 0 views

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    "special education, technology, reading, website, social media, reading activity, writing, sharing, collaboration, publishing, web-based activities, stories, storytelling"
Graeme Wadlow

Shared heritability of attention-deficit/hyperactivity disorder and autism spe... - 0 views

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    SpringerLink - European Child & Adolescent Psychiatry, Volume 19, Number 3
Tero Toivanen

Google-syötteenlukija (4) - 0 views

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    Google had a booth at the Autism Society of America conference last month so they could show off their free software program SketchUp. SketchUp was originally developed for users to create, modify and share 3D models. Turns out SketchUp helps folks with autism develop some skills, too.
Tero Toivanen

Easter Seals and Autism » Blog Archive » What does Google have to do with ... - 0 views

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    Google had a booth at the Autism Society of America conference last month so they could show off their free software program SketchUp. SketchUp was originally developed for users to create, modify and share 3D models. Turns out SketchUp helps folks with autism develop some skills, too.
Graeme Wadlow

Following Gaze: Gaze-Following Behavior as a Window into Social Cognition - 0 views

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    Following Gaze: Gaze-Following Behavior as a Window into Social Cognition
Nicole Mays

The promise of genetics and autism | Psychology Today - 1 views

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    Thanks for sharing!
J B

Tips for Setting up a Classroom for a Child with Autism - 3 views

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    Thanks for sharing, Jeremy!
Tero Toivanen

Autism and early oxygen deprivation 2 | On the Brain by Dr. Mike Merzenich,Ph.D. - 0 views

  • Fraternal twins typically have different placentas, whereas identical twins share a placenta but have different cords. The blood supply, and pre-clamping susceptibility to anoxia, would surely be different.
  • As for the idea that one could statistically detect whether cord clamping is the problem, we can! Amish people do not clamp the cord until placental delivery, and they have no autism rate. The same is true in Somalia, but Somalian immigrants to westernized medical countries have high rates. Try to systematically find out autism rates and immediate cord clamping rates, on a country by country, or region by region basis. It is a task someone should get on immediately, but it will take a lot of effort.
  • An interesting discussion of the “Amish anomaly” re autism incidence has been provided by Dan Olmsted, who went to Amish Country to find the 150 or so individuals there who could be expected to be severely autistic. They aren’t there. He seems pre-disposed to believe that the difference lies with their non-vaccination. Many studies now show that this is unlikely. As David Blake points out, there is another difference in this population: In Amish birthing, by tradition, the cord is not clamped prior to placenta delivery.
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  • The picture with autism in Somali children is a little murkier. It turns out that the incidence of autism is very high in children of Somali origin who were born in the US (several times higher than normal), while it appears to be very low in Somali children born in their native country. Again, vaccination has been identified as the likely cause by Somali parents and by many observers — but again, clamping follows placental delivery in Somalia, while the cord has been clamped without delay as a general practice in Minnesota, where a high incidence of autism in these children of Somali immigrants was first discovered.
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    Comment about the hypothesis that early umbilical cord clamping might contribute to the risk of origin of autism.
Tero Toivanen

New Study: Autism has Multiple Genetic Roots | Suite101 - 3 views

  • The study’s major finding was that children with ASD have significantly more CNVs affecting their genes than children without ASD. Children with ASD have 20 percent more CNVs in general, and 70 percent more CNVs impacting genes known to be associated with ASD or cognitive problems. Significantly, many of the genes that are affected control important functions such as cell proliferation and cell-to-cell communication.
  • Some of the newly discovered genetic variants are inherited, and are found in parents or siblings of children with them. Others, however, seem to have originated spontaneously in the affected child, and do not appear in other family members.
  • While these findings add significantly to the scientific understanding of the genetic and biological underpinnings of ASD, the immediate usefulness is limited. That’s because there are a very large number of CNVs, and each child shows a different pattern of genetic changes. Each of these changes is rare; no CNV showed up in more than one percent of the children studied.
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  • “This will lead to a paradigm shift in understanding the etiology of autism,” says Stephen Scherer, a senior scientist at The Hospital for Sick Children in Toronto, Canada. “Until now, most scientists thought individuals with autism shared common genes. We now think each person has his own rare variations.”
  • If significant CNVs show up, behavioral treatment can be started early. That has been shown to improve children’s outcomes significantly. “If we provide stimulation early, while the brain is still plastic, we can improve cognitive development, social interaction and communication,” says Geri Dawson, Chief Science Officer of Autism Speaks, the major sponsor of the research project.
  • What this new research suggests is that autism and ASD probably result from the interaction between many different genes and a child’s environment. Rather than search for one single cause and one “magic bullet,” researchers will try to find as many significant genetic variants as possible, link them to the biological functions and pathways they control, and then search for medications that can improve or normalize the functioning of damaged pathways.
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