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"Since cancers have individual clonal karyotypes, are immortal and evolve from normal cells treated by carcinogens only after exceedingly long latencies of many months to decades-we deduce that carcinogenesis may be a form of speciation. This theory proposes that carcinogens initiate carcinogenesis by causing aneuploidy, i.e., losses or gains of chromosomes. Aneuploidy destabilizes the karyotype, because it unbalances thousands of collaborating genes including those that synthesize, segregate and repair chromosomes. Driven by this inherent instability aneuploid cells evolve ever-more random karyotypes automatically. Most of these perish, but a very small minority acquires reproductive autonomy-the primary characteristic of cancer cells and species"

Brain tumors are surely frightening but apart from all these painful ways, a brain cancer patient can simply try out the Energy Transmission by Trivedi Masters™.

Come work with us Interested in working with researchers from different disciplines within the Harvard, MIT and Broad community and an unique opportunity to participate in world-class research to make an impact on human health? Come work with us! We are looking for a computational biologists to handle data from a wide variety of experimental methods, focusing on next-gen sequencing technologies. Keep Reading...  SCDE is live The Stem Cell Discovery Engine (SCDE) is an integrated platform that allows users to consistently describe, share and compare cancer and tissue stem cell data. It is made up of an online database of curated experiments coupled to a customized instance of the Galaxy analysis engine with tools for gene list manipulation and molecular profile comparisons. The SCDE currently contains more than 50 stem cell-related experiments. Each has been manually curated and encoded using the ISA-Tab standard to ensure the quality of the data and its annotation. Keep Reading...  The Center for Health Bioinformatics at the Harvard School of Public Health provides consults to researchers for the management, integration and contextual analysis of biological high-throughput data. We are a member of the Center for Stem Cell Bioinformatics, the Environmental Statistics and Bioinformatics Core at the Harvard NIEHS Center for Environmental Health and the Genetics & Bioinformatics Consulting group for Harvard Catalyst and work closely with our colleagues in the Department of Biostatistics and the Program in Quantitative Genomics to act as a single point of contact for computational biology,

A new type of stem cell discovered in the prostate of adult mice can be a source of prostate cancer, according to a new study by researchers

Scientists at the McEwen Centre for Regenerative Medicine and the Ontario Cancer Institute have isolated a human blood stem cell in its purest form: as a single stem cell capable of regenerating the entire blood system. "This discovery means we now have an increasingly detailed road map of the human blood development system, including the much sought after stem cell," says principal investigator John Dick, who holds a Canada Research Chair in Stem Cell Biology and is a Senior Scientist at the McEwen Centre for Regenerative Medicine and the Ontario Cancer Institute, University Health Network (UHN). "We have isolated a single cell that makes all arms of the blood system, which is key to maximizing the potential power of stem cells for use in more clinical applications. Stem cells are so rare that this is a little like finding a needle in a haystack," says Dr. Dick. Ref.: John E. Dick, Isolation of Single Human Hematopoietic Stem Cells Capable of Long-Term Multilineage Engraftment, Science, July 2011: Vol. 333 no. 6039 pp. 218-221 [DOI: 10.1126/science.1201219]

Trivedi Global Inc. Established by Mahendra Kumar Trivedi, this organisation has been doing great contribution to multiple fields like health and wellness, agriculture, metallic science and much more.

We are a nonprofit publisher and advocacy organization. Our mission is to accelerate progress in science and medicine by leading a transformation in research communication. Everything that we publish is open-access - freely available online for anyone to use. Sharing research encourages progress, from protecting the biodiversity of our planet to finding more effective treatments for diseases such as cancer.

Fats in foods like potato chips and french fries make them nearly irresistible because they trigger natural marijuana-like chemicals in the body called endocannabinoids, researchers at the University of California, Irvine, have found. The researchers discovered that when rats tasted something fatty, cells in their upper gut started producing endocannabinoids, while sugars and proteins did not have this effect. How fats create, like, a buzz It starts on the tongue, where fats in food generate a signal that travels first to your brain, and then through a nerve bundle called the vagus to your intestines. There, the signal stimulates the production of endocannabinoids, which initiates a surge in cell signaling that prompts you to totally pig out - probably by initiating the release of digestive chemicals linked to hunger and satiety that compel us to eat more. And that leads to obesity, diabetes and cancer, the researchers said. But they suggest it might be possible to curb this process by obstructing endocannabinoid activity: for example, by using drugs that "clog" cannabinoid receptors. The trick: bypassing the brain to avoid creating anxiety and depression (which happens when endocannabinoid signaling is blocked in the brain). I'm guessing McDonald's won't be adding that drug to their fries. Ref.: Daniele Piomelli, et al., An endocannabinoid signal in the gut controls dietary fat intake, PNAS, 2011; in press

The Trivedi Effect human wellness solutions are medical research for all. Mahendra Trivedi has improved the health of many cancer patients.

Cloud4Cancer Breast Cancer Detection

dass bei Mäusen mindestens 2 der 3 zur Krebstherapie zugelassenen Angiogenese-Hemmer die Bösartigkeit von Tumoren erhöhen können (Cancer Cell 2009, Band 15: Seite 220 und 232). Wurden krebskranke Labortiere mit den Wirkstoffen Sunitinib oder Sorafenib behandelt, verlangsamte sich einerseits das Tumorwachstum deutlich, andererseits drangen die Tumore verstärkt in das umliegende Gewebe ein und bildeten mehr Metastasen. .. Offenbar verlangsamen Angiogenese-Hemmer zunächst das Wachstum eines Tumors, bis dieser auf das knappe Nährstoffangebot reagiert und sich gegen das Aushungern zur Wehr setzt. „Möglicherweise liegt das daran, dass ein Tumor sein Wachstumsverhalten ändert, wenn er nicht mehr ausreichend versorgt wird. Indem er in benachbartes Gewebe eindringt und Tochtergeschwülste bildet, könnte er versuchen, wieder mehr Nachschub zu bekommen

The vision of the GRANATUM project is to bridge the information, knowledge and collaboration gap among biomedical researchers in Europe (at least) ensuring that the biomedical scientific community has homogenized, integrated access to the globally available information and data resources needed to perform complex cancer chemoprevention experiments and conduct studies on large-scale datasets.

Susanna Larsson: Kaffeetrinkerinnen haben ein geringeres Risiko für Hirninfarkte http://www.wissenschaft.de/wissenschaft/news/313141.html (Jingmei Li, Karolinska Institutet, et al.: Breast Cancer Research, Bd. 13, Artikel R49) Yvonne T. van der Schouw (UMC Utrecht) et al.: Arteriosclerosis, Thrombosis, and Vascular Biology: Journal of the American Heart Association, doi: 10.1161/ATVBAHA.109.201939 http://www.wissenschaft.de/wissenschaft/news/311350.html Helena Jernström: Kaffee senkt das Brustkrebsrisiko http://www.wissenschaft.de/wissenschaft/gutzuwissen/291553.html

Mahendra Kumar Trivedi has proven the Trivedi Effect® to be of scientific significance through positive results. The results of the tests were also vouched by eminent scientists and scholars with in depth knowledge in molecular science. During the tests it was proven that when cancer cells bought in contact with the Trivedi Effect® their potency was drastically reduced thus improving the life expectancy and health progress of affected patients.

A collaborative study by researchers at the Salk Institute for Biological Studies uncovered that the tumor suppressor p53 not only stops cells that could become cancerous in their tracks but also controls somatic cell reprogramming.

Scientific communities have clearly indicated that lack of stem cells highly affect the quality of life a person is living. Those who have more stem cells in the body always live the healthy and superior life.

"The unfortunate reality is that a person is likelier build a billion dollar company by writing code than by treating cancer. " (THX James Taylor ‏@JTBiotech ) @Spatzlhirn an @JTBiotech "As with many walks in life, the most glamorous legacies tend to get the most attention and the least are neglected"

It's just plain silly to make claims like this about a basic science paper given that, as I have discussed before, it often takes decades for basic science observations to wend their way through that long strange trip to becoming actual therapies used by clinicians. The life cycle of translational research is long, and efforts to speed it up have only met with mixed success.

Stem cells are generally present in the body of humans and several animals. They divide themselves into some other cells as well with time, which are important for the survival of humans.

"(PhysOrg.com) -- Imagine being able to drop a toothpick on the head of one particular person standing among 100,000 people in a stadium. It sounds impossible, yet this degree of precision at the cellular level has been demonstrated by researchers affiliated with the Johns Hopkins University Institute for NanoBioTechnology. Their study was published online in June in Nature Nanotechnology."