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ScienceDaily (Nov. 3, 2011) - Scientists have recently succeeded in rejuvenating cells from elderly donors (aged over 100). These old cells were reprogrammed in vitro to induced pluripotent stem cells (iPSC) and to rejuvenated and human embryonic stem cells (hESC): cells of all types can again be differentiated after this genuine "rejuvenation" therapy. The results represent significant progress for research into iPSC cells and a further step forwards for regenerative medicine.

To know what the most important virtue of our age is we need to have at least a basic understanding of our age. Our era is becoming increasingly characterized by uncertainty. Fortunately or unfortunately, more than a cursory elucidation of our situation is beyond the scope of this essay. There are geopolitical, economic, technological and environmental trends worth mentioning. When the more philosophical portion of this  discourse arrives I will argue that the virtue of wisdom underlies the meaningfulness and efficacy of all other virtues, and this in broad strokes is primarily due to (1) the aforementioned instability in our surroundings ; (2) the relationship between the deontological and virtue; and (3) the nature of agency itself.  Whether uncertainty itself can provide an ethical foundation for us to elaborate on will be a separate question, and finally I speculate on where wisdom leads us in the context of a philosophy that is politically active and not doomed to irrelevance to and by the larger population.

And this technology is spreading. Immersive Labs, a company in Manhattan, has developed software for digital billboards using cameras to gauge the age range, sex and attention level of a passer-by. The smart signs, scheduled to roll out this month in Los Angeles, San Francisco and New York, deliver ads based on consumers' demographics. In other words, the system is smart enough to display, say, a Gillette ad to a male passer-by rather than an ad for Tampax.

Digital advertising billboards being trialled in Japan are fitted with cameras that read the gender and age group of people looking at them to tailor their commercial messages.

...Historians of science have always had a soft spot for the history of theoretical physics. The great theoretical advances of this century -- relativity and quantum mechanics -- have been documented in fascinating historical accounts that have captivated the mind of the cultivated public. There are no comparable studies of the relations between science and engineering. Breaking with the tradition of the Fachidiot, theoretical physicists have bestowed their romantic autobiographies on the world, portraying themselves as the high priests of the reigning cult.

"In nineteenth‐century America, there was no such person as a "professional scientist." There were professionals and there were scientists, but they were very different. Professionals were men of science who engaged in commercial relations with private enterprises and took fees for their services. Scientists were men of science who rejected such commercial work and feared the corrupting influences of cash and capitalism. Professionals portrayed themselves as active and useful members of an entrepreneurial polity, while scientists styled themselves as crusading reformers, promoters of a purer science and a more research‐oriented university. It was this new ideology, embodied in these new institutions, that spurred these reformers to adopt a special name for themselves-"scientists." One object of this essay, then, is to explain the peculiar Gilded Age, American origins of that ubiquitous term. A larger goal is to explore the different social roles of the professional and the scientist. By attending to the particular vocabulary employed at the time, this essay tries to make clear why a "professional scientist" would have been a contradiction in terms for both the professional and the scientist in nineteenth‐century America. "

Vera Gorbunova and Andrei Seluanov of the University of Rochester have found that human cells undergoing oxidative stress caused by environmental chemicals or routine cellular processes produce a protein (SIRT6) that stimulates cells to repair DNA double-strand breaks, thought to be associated with premature aging and cancer. The team first measured levels of SIRT6 in stressed cells, then treated a second group of stressed cells with a drug that deactivates the protein. DNA repair in the second group stopped, confirming SIRT6′s role.  The team also found that SIRT6 acts in concert with another protein, called PARP1, to make the repairs. Furthermore, the team found that increased levels of SIRT6 lead to faster DNA repair. They suspect that the protein acts as a "master regulator," coordinating stress and DNA repair activities. Ref.: Zhiyong Mao, Christopher Hine, Xiao Tian, Michael Van Meter, Matthew Au, Amita Vaidya, Andrei Seluanov, and Vera Gorbunova. SIRT6 Promotes DNA Repair Under Stress by Activating PARP1. Science 17 June 2011: 1443-1446. [DOI:10.1126/science.1202723]

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