PubMed - Study reports the application of Magnetoencephalography and magnetocardiography in neonates

freepatentsonline - all the inventions of mankind - High-resolution magnetoencephalograph system, components and method

American Academy of Neurology Clinical Neurophysiology Section

PubMed - Abstract - Application of magnetoencephalography (MEG) in epilepsy patients with widespread spike or slow-wave activity

University of Pittsburgh Department of Neurological Surgery - Center for Image-Guided Neurosurgery - Neuromag (MEG) Magnetoencephalography Definition

BMC Pediatrics - Elevated nerve growth factor and neurotrophin-3 levels in cerebrospinal fluid of children with hydrocephalus

PubMed - the biology of neurotrophins, signalling pathways and functional peptide mimetics of neurotrophins and their receptors

PubMed Abstract: Anatomical Society of Great Britian and Ireland 2003 - Neurotrophins and the immuse system

PubMed - Effects of ethanol on the development of circadian time keeping system - Abstract: Ethanol exposure during gestation can have devastating consequences on the developing organism. Children who have a history of prenatally exposure to ethanol may show morphological and functional alterations, referred to as fetal alcohol spectrum disorders (FASD). Fetal alcohol syndrome (FAS), which is characterized by pre- and postnatal growth deficiency, specific cranial/facial features, and dysfunction of central nervous system, is the most severe end of FASD

PubMed Abstract: Environmental enrichment alters neurotrophin levels after fetal alcohol exposure in rats.

PubMed Abstract - Environmental influences on brain neurotrophins in rats

Neurotrophins Definition at RCSB Protein Data Bank

University of Bristol - MRC Centre for Synaptic Plasticity - Glutamate Receptors - Glutamate is the major excitatory neurotransmitter in central nervous system (CNS) and as such the glutamate receptors play a vital role in the mediation of excitatory synaptic transmission -

In the new study, researchers led by Maiken Nedergaard, M.D., D.MSc., of the University of Rochester , showed that glutamate produced by astrocytes can trigger seizures.

"The focus of the study is on the plentiful neurons that communicate with each other through the neurotransmitter glutamate. While glutamate is a necessary excitatory substance in the nervous system, in excess, it overstimulates and becomes toxic - excitotoxic - to neurons. Fortunately, neighboring astrocytes can mop up the excess via molecular transporters embedded in their outer membranes. The chief transporter is a protein called EAAT2. Earlier Rothstein's group showed that astroglia - and their EAAT2 protein - are critical for normal neuron activity. In test rats whose astroglia lack the EAAT2 equivalent there's not only a flood of toxic glutamate but a resulting neuron death that leads to paralysis."

The Solomon H. Snyder Department of Neuroscience - the Johns Hopkins University - Asst Professor Mary Ann wilson, PhD - Mechanisms of Development, Plasticity and Injury in the Immature Brain

NINDS - what's Old is New Again - Antibiotic Protects Nerves By Removing Excess Glutamate

Cold spring Harbor Laboratory's Dolan DNA Learning Center - Harlem DNA Lab & DNA Learning Center West - Glutamate Damage and Stroke - Glutmate Description, transcript - 3D pdf reference and information source

NINDS - Receptor Biology Section - Division of Intramural Research - Labs at NINDS - Glutamate

NINDS - Researchers Identify a Signal for Cell Death during Stroke - In a new study, researchers have identified a signal that promotes the death of vulnerable brain cells in an animal model of stroke. In the future, drugs designed to inhibit this death signal might help reduce brain damage in stroke patients