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Contents contributed and discussions participated by orasa sukmark

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orasa sukmark

Making a Friendlier Mosquito - Biology Online - 1 views

www.biology-online.org/...aking_friendlier_mosquito.html

making mosquito biology Genes

shared by orasa sukmark on 16 Apr 12 - No Cached
  • Genetically modified mosquitoes that cannot transmit malaria are one hope for battling the disease that still kills over one million people a year. But that plan faces some serious snags, according to UC Davis researchers who are suggesting an alternative strategy.
  • nsmit malaria are one hope for battling the disease that still kills over one million people a year. But that plan face
  • Genetically modified mosquitoes that cannot transmit malaria are one hope for battling the disease that still kills over one million people a year.
  • ...7 more annotations...
  • enetically modified mosquitoes that cannot transmit malaria are one hope for battling the disease that still kills over one million people a year. But that plan faces some serious snags, according to UC Davis researchers who are suggesting an alternative strategy
  • releasing into the wild mosquitoes genetically engineered to resist malaria
  • If the resistant mosquitoes breed and spread their genes through the population, malaria transmission should be shut down.
  • the malaria resistance genes available are not very effective
  • , there's no way to reliably push the genes through the population.
  • Transposons are essentially DNA parasites that snip themselves in or out of the genome under the right circumstances. Scientists can add a new gene into a transposon and use it to carry that DNA into the insect genome. But it's in the interest of that transposon to just get rid of the extra DNA,
  • a transposon that gives an advantage to mosquitoes that already carry genes to block malaria, so that those genes spread through the population by natural selection.
  • orasa sukmark on 16 Apr 12
     
    scientists have found a way to reduce mosquitoes that can transmit malaria. 
orasa sukmark

Cloned Meat & Milk Coming - 1 views

www.i-sis.org.uk/loned_Meat_and_Milk_Coming.php

meat milk coming Genes

shared by orasa sukmark on 16 Apr 12 - No Cached
  • Milk and meat from ‘cloned’ animals’ offspring could soon be on sale without any labels
  • The UK government has no objection to selling meat and milk from clones.
  • There are currently just over 100 offspring of cloned animals on British farms, but this will rise dramatically if the EC gets its way.
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  • According to BEUC, an overwhelming majority of EU consumers do not want cloning to be used for food production purposes. Some 84 per cent are concerned about the long-term health and safety impacts.
  • orasa sukmark on 16 Apr 12
     
    Cloned Meat and milk will be sold soon.
orasa sukmark

Shot of Young Stem Cells Makes Rapidly Aging Mice Live Much Longer and Healthier - Gate... - 1 views

www.gate2biotech.com/-2130

shot stem cells aging much mice longer gate biotech com DNA genetics disease gene RNA brain human epigenetics research Genes

shared by orasa sukmark on 16 Apr 12 - No Cached
  • animals that got the stem/progenitor cells improved their health and lived two to three times longer than expected,
  • "Our experiments showed that mice that have progeria, a disorder of premature aging, were healthier and lived longer after an injection of stem cells from young, healthy animals," Dr. Niedernhofer said. "That tells us that stem cell dysfunction is a cause of the changes we see with aging."
  • "Typically the progeria mice die at around 21 to 28 days of age, but the treated animals lived far longer -- some even lived beyond 66 days. They also were in better general health."
  • ...2 more annotations...
  • we injected stem/progenitor cells from young, healthy mice into the abdomens of 17-day-old progeria mice,
  • As the progeria mice age, they lose muscle mass in their hind limbs, hunch over, tremble, and move slowly and awkwardly. Affected mice that got a shot of stem cells just before showing the first signs of aging were more like normal mice, and they grew almost as large.
  • orasa sukmark on 16 Apr 12
     
    the experiment from the University of Pittsburgh shows that the mice can be stronger live longer after they were injected with stem cells from young healthy animals.
orasa sukmark

Genetic adaptation of fat metabolism key to development of human brain - 0 views

www.sciencedaily.com/...120412133056.htm

adaptation fat metabolism key development human brain

shared by orasa sukmark on 15 Apr 12 - No Cached
  • orasa sukmark on 15 Apr 12
     
    Genetic adaptation for high production of Omega-3 and Omega-6 fatty acids is found only in humans can show genetic adaptation of human fat metabolism.
orasa sukmark

Gene therapy - 0 views

www.sciencedaily.com/...gene_therapy.htm

gene therapy

shared by orasa sukmark on 02 Apr 12 - No Cached
  • orasa sukmark on 02 Apr 12
     
    general info on gene therapy
orasa sukmark

Junk DNA Can Revive and Cause Disease, Study Finds - NYTimes.com - 0 views

www.nytimes.com/...20gene.html

shared by orasa sukmark on 30 Mar 12 - No Cached
  • a dead gene come back to life and cause a disease.
  • Some of those genes, surprised geneticists reported Thursday, can rise from the dead like zombies, waking up to cause one of the most common forms of muscular dystrophy.
  • FSHD, is one of the most common forms of muscular dystrophy.
  • ...4 more annotations...
  • in a way FSHD was the easy case — it is a disease that affects every single person who inherits the genetic defect. Other diseases are more subtle, affecting some people more than others, causing a range of symptoms.
  • The dead gene was also repeated on chromosome 10, but that area of repeats seemed innocuous, unrelated to the disease. Only chromosome 4 was a problem.
  • But the transcriptions were faulty, disintegrating right away. They were missing a crucial section, called a poly (A) sequence, needed to stabilize them.
  • extra copies change the chromosome’s structure, shutting off the whole region so it cannot be used.
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