Skip to main content

Home/ TOK@ISPrague/ Group items tagged obesity

Rss Feed Group items tagged

Filter: All | Bookmarks | Topics Simple Middle
markfrankel18

Does This Ad Make Me Fat? - NYTimes.com - 1 views

www.nytimes.com/...does-this-ad-make-me-fat.html

science knowledge reason

shared by markfrankel18 on 10 Mar 13 - No Cached
  • A team of researchers walked every street in 228 census tracts around Los Angeles and New Orleans and recorded every outdoor ad they saw. Another group surveyed 2,881 residents of the same census tracts by telephone, paying them to report their height, weight and other information. After analyzing this hard-won data, the authors conclude: “For every 10 percent increase in food advertisements, the odds of being obese increased by 5 percent.” That is, areas with more outdoor food ads have a higher proportion of obese people than ones with fewer ads.
  • The problem is that their policy recommendations rest on a crucial but unjustified assumption: that any link between obesity and advertising occurs because more advertising causes higher rates of obesity. But the study at hand showed only an association: people living in areas with more food ads were more likely to be obese than people living in areas with fewer food ads. To be fair, the researchers correctly note that additional steps would be needed to prove that food ads cause obesity. But until those steps are taken, talk of restricting ads is premature. In fact, it is easy to imagine how the causation could run the opposite way (something the article did not mention): If food vendors believe obese people are more likely than non-obese people to buy their products, they will place more ads in areas where obese people already live. Suppose we counted ads for fitness-oriented products like bicycles and bottled water, and found more of those ads in places with less obesity. Would it then be wise anti-obesity policy to subsidize such ads? Or would the smarter conclusion be that the fitness companies suspect that the obese are less likely than the fit to buy their products?
  • When we seek to base policy on evidence, we must remember that not all “evidence” is created equal. Taken at face value, the study on ads and obesity provides some indication that the two are linked, but no evidence that food ads cause obesity. The fact that the causal conclusion may coincide with a moral belief — that it is wrong to tempt people who overeat by showing them ads for food — does not make it valid.
Lawrence Hrubes

Same but Different - The New Yorker - 0 views

www.newyorker.com/...breakthroughs-in-epigenetics

science biology genetics nature DNA ethics

shared by Lawrence Hrubes on 26 Apr 16 - No Cached
  • Why are identical twins alike? In the late nineteen-seventies, a team of scientists in Minnesota set out to determine how much these similarities arose from genes, rather than environments—from “nature,” rather than “nurture.” Scouring thousands of adoption records and news clips, the researchers gleaned a rare cohort of fifty-six identical twins who had been separated at birth. Reared in different families and different cities, often in vastly dissimilar circumstances, these twins shared only their genomes. Yet on tests designed to measure personality, attitudes, temperaments, and anxieties, they converged astonishingly. Social and political attitudes were powerfully correlated: liberals clustered with liberals, and orthodoxy was twinned with orthodoxy. The same went for religiosity (or its absence), even for the ability to be transported by an aesthetic experience. Two brothers, separated by geographic and economic continents, might be brought to tears by the same Chopin nocturne, as if responding to some subtle, common chord struck by their genomes.
  • It’s one thing to study epigenetic changes across the life of a single organism, or down a line of cells. The more tantalizing question is whether epigenetic messages can, like genes, cross from parents to their offspring.
  • The most suggestive evidence for such transgenerational transmission may come from a macabre human experiment. In September, 1944, amid the most vengeful phase of the Second World War, German troops occupying the Netherlands banned the export of food and coal to its northern parts. Acute famine followed, called the Hongerwinter—the hunger winter. Tens of thousands of men, women, and children died of malnourishment; millions suffered it and survived. Not surprisingly, the children who endured the Hongerwinter experienced chronic health issues. In the nineteen-eighties, however, a curious pattern emerged: when the children born to women who were pregnant during the famine grew up, they had higher rates of morbidity as well—including obesity, diabetes, and mental illness. (Malnourishment in utero can cause the body to sequester higher amounts of fat in order to protect itself from caloric loss.) Methylation alterations were also seen in regions of their DNA associated with growth and development. But the oddest result didn’t emerge for another generation. A decade ago, when the grandchildren of men and women exposed to the famine were studied, they, too, were reported to have had higher rates of illness. (These findings have been challenged, and research into this cohort continues.) “Genes cannot change in an entire population in just two generations,” Allis said. “But some memory of metabolic stress could have become heritable.”
Lawrence Hrubes

Why Do We Eat, and Why Do We Gain Weight? - www.newyorker.com - 2 views

www.readability.com/...doxpy7i3

science biology obesity food hunger psychology TOK

shared by Lawrence Hrubes on 13 Apr 14 - No Cached
  • Here are a few of the things that can make you hungry: seeing, smelling, reading, or even thinking about food. Hearing music that reminds you of a good meal. Walking by a place where you once ate something good. Even after you’ve just had a hearty lunch, imagining something delicious can make you salivate. Being genuinely hungry, on the other hand—in the sense of physiologically needing food—matters little. It’s enough to walk by a doughnut shop to start wanting a doughnut. Studies show that rats that have eaten a lot are just as eager to eat chocolate cereal as hungry rats are to eat laboratory chow. Humans don’t seem all that different. More often than not, we eat because we want to eat—not because we need to. Recent studies show that our physical level of hunger, in fact, does not correlate strongly with how much hunger we say that we feel or how much food we go on to consume. That’s something of a departure from commonly held views of what it means to be hungry.
Lawrence Hrubes

Walter Mischel, The Marshallow Test, and Self-Control - The New Yorker - 1 views

www.newyorker.com/...hologist-studying-self-control

TOK psychology psychiatry self-control gratification delay social studies human sciences

shared by Lawrence Hrubes on 14 Feb 15 - No Cached
  • Mischel’s story isn’t surprising—nicotine is addictive, and quitting is difficult—except for one thing: Mischel is the creator of the marshmallow test, one of the most famous experiments in the history of psychology, which is often cited as evidence of the importance of self-control. In the original test, which was administered at the Bing Nursery School, at Stanford, in the nineteen-sixties, Mischel’s team would present a child with a treat (marshmallows were just one option) and tell her that she could either eat the one treat immediately or wait alone in the room for several minutes until the researcher returned, at which point she could have two treats. The promised treats were always visible and the child knew that all she had to do to stop the agonizing wait was ring a bell to call the experimenter back—although in that case, she wouldn’t get the second treat. The longer a child delayed gratification, Mischel found—that is, the longer she was able to wait—the better she would fare later in life at numerous measures of what we now call executive function. She would perform better academically, earn more money, and be healthier and happier. She would also be more likely to avoid a number of negative outcomes, including jail time, obesity, and drug use.
  • It was not until one day in the late nineteen-sixties, when he saw a man with metastasized lung cancer in the halls of Stanford’s medical school—chest exposed, head shaved, little green “x” marks all over his body, marking the points where radiation would go—that Mischel realized he was fooling himself. Finally, something clicked. From then on, each time he wanted a cigarette (approximately every three minutes, by his count) he would create a picture in his mind of the man in the hallway. As he described it to me, “I changed the objective value of the cigarette. It went from something I craved to something disgusting.” He hasn’t had a smoke since.
  • Lawrence Hrubes on 14 Feb 15
     
    "Mischel, who is now eighty-four years old, has just published his first popular book, "The Marshmallow Test: Mastering Self-Control." It is part memoir, part scientific analysis, and part self-help guide. In the book, he describes the original impetus for the marshmallow study. At the time, his daughters, Judith, Rebecca, and Linda, were three, four, and five years old, respectively. "I began to see this fascinating phenomenon where they morphed from being highly impulsive, immediate creatures who couldn't delay anything," he told me. "There were these amazingly rapid changes-everything around them was the same, but something inside them had changed. I realized I didn't have a clue what was going on in their heads." He wondered what was it that had enabled them to go from deciding that they wanted to wait to actually being able to do so. He found the answer among their classmates at the Bing preschool."
Lawrence Hrubes

The Excrement Experiment - The New Yorker - 0 views

www.newyorker.com/...excrement-experiment

science experiment research biology

shared by Lawrence Hrubes on 29 Feb 16 - No Cached
  • Crohn’s disease affects as many as seven hundred thousand Americans, but, like other autoimmune disorders, it remains poorly understood and is considered incurable. (Autoimmune disorders are thought to arise when the immune system attacks healthy tissue, mistaking it for a threat.) The standard treatments for Crohn’s often don’t work, or work only temporarily, and many have serious side effects.
  • His mother showed him an article from the Times about a man who had been nearly bedridden by ulcerative colitis—a condition related to Crohn’s—and who had largely recovered after a month or so of fecal transplants. Gravel found a how-to book on Amazon and bought the recommended equipment: a blender, a rectal syringe, saline solution, surgical gloves, Tupperware containers. His wife agreed to be his donor.
  • New research suggests that the microbes in our guts—and, consequently, in our stool—may play a role in conditions ranging from autoimmune disorders to allergies and obesity, and reports of recoveries by patients who, with or without the help of doctors, have received these bacteria-rich infusions have spurred demand for the procedure. A year and a half ago, a few dozen physicians in the United States offered FMT. Today, hundreds do, and OpenBiome, a nonprofit stool bank founded last year by graduate students at M.I.T., ships more than fifty specimens each week to hospitals in thirty-six states. The Cleveland Clinic named fecal transplantation one of the top ten medical innovations for 2014, and biotech companies are competing to put stool-based therapies through clinical trials and onto the market. In medicine, at any rate, human excrement has become a precious commodity.
1 - 5 of 5
Showing 20▼ items per page