which is a major reason for the regaining of lost weight with dieting as well being the mechanism behind stress induced weight gain (it is not due to increased cortisol).
f greater than 10, it demonstrates there is a degree of leptin resistance contributing to an inability to lose weight
that it is difficult to lose weight with leptin resistance. High carbohydrate diets and in particular high-fructose corn syrup is shown to significantly increase leptin resistance and is a likely mechanism that high fructose corn syrup is associated with obesity
it is problem inside the cell that the inactive T4 is not converted to T3 but rather to a mirror image of T3 called reverse T3. The reverse T3 has the opposite effect of T3, blocking the effects of T3 and lowering rather than increasing metabolism.
Studies are showing that stress and dieting (especially yo-yo dieting) can set this hormone into action as well as chronic illness such as diabetes, chronic fatigue syndrome and fibromyalgia.
As soon as the body senses a reduction in calories, the production of reverse T3 is stimulated to lower metabolism
With chronic dieting or stress, the body often stays in this "starvation mode" with elevated levels of reverse T3 and decreased levels of T3, which is a major reason for the regaining of lost weight with dieting as well being the mechanism behind stress induced weight gain (it is not due to increased cortisol).
which is a major reason for the regaining of lost weight with dieting as well being the mechanism behind stress induced weight gain (it is not due to increased cortisol).
which is a major reason for the regaining of lost weight with dieting as well being the mechanism behind stress induced weight gain (it is not due to increased cortisol).
Studies are showing that such standard testing will miss 80% of thyroid dysfunction
women with the polycystic ovary syndrome, D-chiro-inositol reduces circulating insulin, decreases serum androgens, and ameliorates some of the metabolic abnormalities (increased blood pressure and hypertriglyceridemia) of syndrome X.
These data also suggest that endogenous progesterone could play a modulation role on pituitary hormone secretion, stimulating GH and PRL release and enhancing the inhibitory action of sleep on TSH secretion.
normally cycling young women, daytime GH and PRL secretions are increased in luteal phase
aromatase inhibition in men to increase Testosterone:Estradiol ratio. Men with infertility have a low Testosterone:Estradiol ratio. Aromatase inhibitors inhibit Testosterone to Estradiol conversion
Approximately 80% of women reported overall satisfaction with the micronized progesterone-containing regimen. A micronized progesterone-containing HRT regimen offers the potential for improved QOL as measured by improvement of menopause-associated symptoms.
salivary cortisol were studied by Russian cosmonauts on board orbital station Mir during
long-term space flights (SF)
After the long-term SF, individual ratios of salivary and plasma cortisol levels increased against the background
of increased plasma content of the hormone, i.e., the fraction of free, physiologically active hormone in the total pool of
circulating molecules decreased.
salivary testing of estradiol and progesterone shown to be statistically similar to blood estradiol and progesterone. And, "salivary sampling is simple, convenient, and stress free". End of debate.
Prenatal exposure to BPA has been shown to alter a variety of reproductive endocrine parameters, such as testosterone and luteinizing hormone levels
arly onset of sexual maturation of female mice
imbalanced T-helper (TH)1/TH2 immune responses have been demonstrated on exposure to BPA
indicating that BPA exerted its effects by reducing the number of Treg cells.
Exposure to BPA by subcutaneous injection in adulthood significantly promoted antigen-stimulated production of IL-4, IL-10, and IL-13 in TH2-skewed
BPA can leak from the placenta and accumulate in the fetus
We showed that prenatal exposure to BPA increased the production of a TH1 cytokine, IFN-γ, and a TH2 cytokine, IL-4, after the offspring developed, suggesting that prenatal exposure to BPA can induce persistent immunologic effects lasting into adulthood.
These results are consistent with a previous report that fetal exposure to BPA augmented TH1 and TH2 immune responses
our results clearly demonstrate that the production of TH2 cytokines is promoted by BPA in adult mice and in offspring during developmental exposure.
The decrease of Treg cells would predispose to immune dysfunction in aged individuals, explaining their higher risk of immune-mediated diseases, cancer, and infections.
BPA might cause these diseases. Thus, avoiding exposure to or promoting the excretion of BPA and other EDCs would help in preventing diseases and adverse health effects.
This study looked at Total serum Testosterone and lipid profiles in men with CAD. What they found is that serum Total Testosterone is positively associated with HDL and negatively associated with LDL.
Small study of hypogondal men at mean age of 37 years finds low T associated with increased TNF-alpha and MIP. The Testosterone was evaluated as serum Total testosterone and serum calculated free Testosterone.
Study of Japanese men > 30 found that lower serum TT was associated with an increased OR of metabolic syndrome. The men in this study were "healthy". This study looked at TT as a means to help predict MetS.